Parimal Chowdhury
University of Arkansas for Medical Sciences
114 Papers
1.1K Citations
Parimal Chowdhury is an academic researcher from University of Arkansas for Medical Sciences. The author has contributed to research in topics: Cholecystokinin & Nicotine. The author has an hindex of 23, co-authored 114 publications. Previous affiliations of Parimal Chowdhury include Rutgers University & Kyoto University.
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Papers
•Journal Article
Lipid peroxidation in rat brain is increased by simulated weightlessness and decreased by a soy-protein diet.
TL;DR: The mean levels of lipid peroxidation products in brain tissues were decreased in the rats fed the soy diet and were increased in the Rats that were tail-suspended to simulate weightlessness, when compared to those of rats fed a regular diet.
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Pathophysiological effects of nicotine on the pancreas: an update.
TL;DR: Exposure of laboratory animals to nicotine clearly supports the notion that nicotine can induce pancreatic injury, and a possible pathogenetic mechanism of nicotine in the pancreas activating multiple signal transduction pathways is schematically summarized in Figure 1.
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Pathophysiology of alcoholic pancreatitis: an overview.
Parimal Chowdhury,Priya Gupta +1 more
TL;DR: The current review cites the updated literature on the alcohol metabolism, its effects on gastrointestinal and pancreatic function and in causing pancreatic injury, genetic predisposition of alcohol induced pancreatitis.
74
Metabolic and pathologic effects of nicotine on gastrointestinal tract and pancreas of rats
TL;DR: The data suggest that nicotine ingested chronically alters metabolic, endocrine, and pathologic factors that may be responsible for the development of gastrointestinal ulcers and pancreatitis, which appear to have been substantially prevented by abstinence of nicotine ingestion.
63
Smoking and pancreatic disorders.
TL;DR: A hypothetical pancreatic acinar cell model showing multiple signal transduction pathways is presented relating to possible mechanisms of action of nicotine on the pathophysiology of the pancreas.
56