Paolo Fabbri
University of Florence
235 Papers
1.2K Citations
Paolo Fabbri is an academic researcher from University of Florence. The author has contributed to research in topics: Medicine & Dermatitis herpetiformis. The author has an hindex of 38, co-authored 230 publications. Previous affiliations of Paolo Fabbri include UniFi & University of Modena and Reggio Emilia.
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Papers
•Journal Article
International survey on the management of patients with SLE. I. General data on the participating centers and the results of a questionnaire regarding mucocutaneous involvement
Claudio Vitali,Andrea Doria,Angela Tincani,Paolo Fabbri,G. Balestrieri,Mauro Galeazzi,Pl Meroni,Paola Migliorini,Rossella Neri,Antonio Tavoni,Stefano Bombardieri +10 more
TL;DR: The present survey indicates that, although most of the participating centres had extensive experience in the management of SLE, their approach to the MC manifestations was not homogeneous, and collaborative studies are clearly needed, particularly to optimise the therapeutic protocols.
IL-4, IL-5, TGF-beta1 and IFN-gamma mRNAs detected by a new in situ amplification system in cicatricial pemphigoid.
TL;DR: The results suggest the presence of a T‐cell population with a mixed cytokine pattern in the cellular infiltrate of both blistering diseases, with a corresponding increase of Th2‐like activity in fully developed lesions, irrespective of the different sites involved.
Chronic idiopathic urticaria: infiltrating cells and related cytokines in autologous serum-induced wheals.
Marzia Caproni,Barbara Giomi,Walter Volpi,Lucilla Melani,E. Schincaglia,D. Macchia,Mariangela Manfredi,Alberino D'Agata,Paolo Fabbri +8 more
TL;DR: The data show that the immunoinflammatory features of ASST-induced wheals involve a prevalent role of lymphocytes (with a mixed Th1/Th2 response), with strong neutrophil infiltration and activity and involvement of the chemokine pathway.
Cytokine profile and supposed contribution to scarring in cicatricial pemphigoid
Marzia Caproni,Anna Calzolari,E. Salvatore,Barbara Giomi,Walter Volpi,Alberino D'Agata,Marco Santucci,Paolo Fabbri +7 more
TL;DR: The presence of a mixed cytokine pattern in the cellular infiltrate of both blistering diseases, with a corresponding increase of Th2-like activity in fully developed lesions, irrespective of the different sites involved, highlights the involvement of additional factors in the scarring course typical of CP.