Pablo Valle-Leija
National Autonomous University of Mexico
8 Papers
16 Citations
Pablo Valle-Leija is an academic researcher from National Autonomous University of Mexico. The author has contributed to research in topics: Nerve growth factor & Superior cervical ganglion. The author has an hindex of 6, co-authored 8 publications. Previous affiliations of Pablo Valle-Leija include McGill University.
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Papers
Odor memory stability after reinnervation of the olfactory bulb.
Eduardo Blanco-Hernández,Pablo Valle-Leija,Viviana Zomosa-Signoret,René Drucker-Colín,Román Vidaltamayo +4 more
TL;DR: The results show that 45 days after methimazole-induced lesion, axonal projections to the bulb of M72 and I7 populations are largely reestablished and regenerated glomeruli are re-formed within the same areas as those of control, unexposed mice.
Supernumerary Formation of Olfactory Glomeruli Induced by Chronic Odorant Exposure: A Constructivist Expression of Neural Plasticity
Pablo Valle-Leija,Eduardo Blanco-Hernández,René Drucker-Colín,Gabriel Gutiérrez-Ospina,Román Vidaltamayo +4 more
TL;DR: Overall, the results support that sensory experience indeed instructs the permanent formation of specific glomeruli in the mouse olfactory bulb by means of constructivist processes.
Maternal hyperthyroidism in rats impairs stress coping of adult offspring.
Li-Mei Zhang,Vito S. Hernández,Mauricio Medina-Pizarro,Pablo Valle-Leija,Arturo Vega-González,Teresa Morales +5 more
TL;DR: The results suggest that MH in rats leads to the offspring being more vulnerable to stress in adulthood, and impaired spatial learning after acute and chronic stress.
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Odorant Receptors Signaling Instructs the Development and Plasticity of the Glomerular Map
TL;DR: The olfactory system provides a great opportunity to explore the mechanisms that underlie the formation and function of neural circuits because of the simplicity of its structure, and essential information about the molecular basis of these processes is addressed.
Unilateral olfactory deficit in a hemiparkinson's disease mouse model.
TL;DR: It is proposed that the olfactory deficit was caused by dopaminergic denervation to the Olfactory tubercle and nucleus accumbens of Parkinson’s disease.
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