Norman E. Miller
St Thomas' Hospital
16 Papers
510 Citations
Norman E. Miller is an academic researcher from St Thomas' Hospital. The author has contributed to research in topics: Cholesterol & Lipoprotein. The author has an hindex of 12, co-authored 16 publications. Previous affiliations of Norman E. Miller include University of Tromsø & Toronto Western Hospital.
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Papers
Sex, plasma lipoproteins, and atherosclerosis: Prevailing assumptions and outstanding questions
TL;DR: The hypothesis that the incidence of coronary heart disease (CHD) is higher in men than in women due to differences in plasma lipoprotein risk factors between the sexes is reviewed and evidence relating these sex differences in CHD and lipoproteins to the effects of sex hormones is critically examined.
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Association between dietary fat intake and plasma factor VII coagulant activity. A predictor of cardiovascular mortality
TL;DR: The character of the VIIc response to fat intake suggested an association with post-prandial lipaemia, and a high fat intake may lead not only to coronary atheroma but also to fibrin deposition and thrombus formation through direct activation of the coagulation system.
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Pathophysiology of Male Hypogonadism Associated with Endogenous Hyperestrogenism — Evidence for Dual Defects in the Gonadal Axis
Johannes D. Veldhuis,James R. Sowers,Alan D. Rogol,Frederick A. Klein,Norman E. Miller,Maria L. Dufau +5 more
TL;DR: It is necessary to select patients suitable for vaginal or laparoscopic mesh placement for surgery on the basis of prior history and once they provide informed consent for surgery.
A longitudinal study of the biological variability of plasma lipoproteins in healthy young adults.
O.D. Mjøs,O.D. Mjøs,S. N. Rao,S. N. Rao,L. Bjøru,L. Bjøru,T. Henden,T. Henden,D. S. Thelle,D. S. Thelle,O. H. Førde,O. H. Førde,Norman E. Miller,Norman E. Miller +13 more
TL;DR: Fluctuations of HDL-C with time appear to be related in part to variations in triglyceride-rich lipoprotein metabolism, which needs to be taken into account when making comparisons in epidemiological studies of the predictive powers of single on-entry measurements for future disease.
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Variations in the apolipoprotein AI-CIII-AIV gene region and in lecithin:cholesterol acyltransferase concentration are determinants of plasma cholesterol concentrations.
TL;DR: Variation in this gene region may be one of the polygenetic factors involved in determining cholesterol levels in the normal population, and associations of the RFLPs with plasma cholesterol concentration may reflect changes in LCAT activity secondary to qualitative or quantitative changes in one or more of these apolipoproteins.
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