Noriko Funato
Tokyo Medical and Dental University
33 Papers
341 Citations
Noriko Funato is an academic researcher from Tokyo Medical and Dental University. The author has contributed to research in topics: Biology & Cellular differentiation. The author has an hindex of 16, co-authored 30 publications. Previous affiliations of Noriko Funato include University of Texas Southwestern Medical Center.
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Papers
Common Regulation of Growth Arrest and Differentiation of Osteoblasts by Helix-Loop-Helix Factors
TL;DR: The results demonstrate that a common regulatory system is involved in at least two distinct steps in osteoblastic differentiation and provide the molecular basis of Saethre-Chotzen syndrome, caused by mutations of the TWISTand FGFR3 genes.
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Hand transcription factors cooperatively regulate development of the distal midline mesenchyme.
Ana C. Barbosa,Noriko Funato,Shelby L. Chapman,Marc D. McKee,James A. Richardson,Eric N. Olson,Hiromi Yanagisawa +6 more
TL;DR: The importance of Hand transcription factors in the transcriptional circuitry of craniofacial and tooth development is demonstrated as well as the early embryonic lethality of Hand1 or Hand2-null embryos.
73
Hand2 controls osteoblast differentiation in the branchial arch by inhibiting DNA binding of Runx2
Noriko Funato,Shelby L. Chapman,Marc D. McKee,Hiromasa Funato,Jesse Morris,John M. Shelton,James A. Richardson,Hiromi Yanagisawa +7 more
TL;DR: It is shown that Hand2 acts as a novel inhibitor of the Runx2-DNA interaction and thereby regulates osteoblast differentiation in branchial arch development.
Basic helix-loop-helix transcription factor epicardin/capsulin/Pod-1 suppresses differentiation by negative regulation of transcription.
TL;DR: Results indicate that epicardin/capsulin/Pod-1 functions as a negative regulator of differentiation of myoblasts through transcription in at least two distinct steps, cell growth arrest and lineage-specific differentiation.
64
Basic fibroblast growth factor induces apoptosis in myofibroblastic cells isolated from rat palatal mucosa
TL;DR: Findings suggested that bFGF may be a potential stimulator of apoptosis in myofibroblasts during palatal scar formation and that FGFR may be responsible for this process.
46