Nicolas Clere
University of Angers
87 Papers
261 Citations
Nicolas Clere is an academic researcher from University of Angers. The author has contributed to research in topics: Angiogenesis & Tumor microenvironment. The author has an hindex of 15, co-authored 83 publications. Previous affiliations of Nicolas Clere include French Institute of Health and Medical Research & University of Franche-Comté.
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Papers
Epigallocatechin Gallate: A Review of Its Beneficial Properties to Prevent Metabolic Syndrome.
TL;DR: According to in vitro and in vivo model data, this review attempts to increase the understanding about the beneficial properties of EGCG to prevent metabolic syndrome.
Endothelial-to-Mesenchymal Transition in Cancer.
TL;DR: This review highlights the process of EndMT in the context of cancer and discusses it as an important adaptive process of the tumor microenvironment that favors tumor growth and dissemination but also resistance to treatment.
Vasculogenic mimicry, a complex and devious process favoring tumorigenesis - Interest in making it a therapeutic target.
TL;DR: In this article, the authors discuss the importance of VM-associated mediators in antitumor therapy and how it could allow to better understand the resistance to anticancer therapy and highlight the predominant molecular targets and signaling pathways involved.
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Endothelial-to-mesenchymal transition (EndoMT): roles in tumorigenesis, metastatic extravasation and therapy resistance.
TL;DR: The role of EndoMT in tumorigenesis, metastasis, and its potential implication in cancer therapy resistance is discussed and its main induction pathways are summarized.
Deficiency or blockade of angiotensin II type 2 receptor delays tumorigenesis by inhibiting malignant cell proliferation and angiogenesis
Nicolas Clere,Isabelle Corre,Sébastien Faure,Anne-Laure Guihot,Emilie Vessieres,Marie Chalopin,Alain Morel,Olivier Coqueret,Lutz Hein,Yves Delneste,François Paris,Daniel Henrion +11 more
TL;DR: Novel mechanisms by which AT2R would promote tumor development, favoring both malignant cell proliferation and tumor angiogenesis are uncovered, particularly in LL/2 and 3‐MCA tumor cells.
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