Navtej S. Buttar
Mayo Clinic
228 Papers
1K Citations
Navtej S. Buttar is an academic researcher from Mayo Clinic. The author has contributed to research in topics: Barrett's esophagus & Medicine. The author has an hindex of 39, co-authored 179 publications. Previous affiliations of Navtej S. Buttar include University of Rochester.
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Papers
Direct Percutaneous Endoscopic Jejunostomy: Outcomes in 307 Consecutive Attempts
John T. Maple,Bret T. Petersen,Todd H. Baron,Christopher J. Gostout,Louis M. Wong Kee Song,Navtej S. Buttar +5 more
TL;DR: DPEJ is a useful technique to gain enteral access that obviates the need for surgery and is more reliable than percutaneous gastrostomy with jejunal extension, patients and physicians should be aware of the risks involved.
187
ASGE guideline for infection control during GI endoscopy.
Audrey H. Calderwood,Lukejohn W. Day,V. Raman Muthusamy,James Collins,Ralph David Hambrick,Andrew Brock,Nalini M. Guda,Jonathan M. Buscaglia,Bret T. Petersen,Navtej S. Buttar,Lauren G. Khanna,Vladimir Kushnir,Aparna Repaka,Nicolas Villa,Glenn M. Eisen +14 more
TL;DR: This guideline is intended to be an educational tool to provide information that may assist endoscopists in delivering care to patients and should not be construed as establishing a legal standard of care or as encouraging, advocating, requiring, or discouraging any particular treatment.
180
Bone morphogenetic protein 4 expressed in esophagitis induces a columnar phenotype in esophageal squamous cells.
Francesca Milano,Jantine W. van Baal,Navtej S. Buttar,Agnieszka M. Rygiel,Floor de Kort,Cathrine J. DeMars,Wilda D. Rosmolen,Jacques J. Bergman,Jan van Marle,Kenneth K. Wang,Maikel P. Peppelenbosch,Kausilia K. Krishnadath +11 more
TL;DR: It is suggested that the BMP pathway could play a role in the transformation of normal esophageal squamous cells into columnar cells.
174
The effect of selective cyclooxygenase-2 inhibition in Barrett's esophagus epithelium: an in vitro study.
Navtej S. Buttar,Kenneth K. Wang,Marlys Anderson,Ross A. Dierkhising,Rodney J. Pacifico,Krishnawatie K. Krishnadath,Lori S. Lutzke +6 more
TL;DR: COx-2 is functionally active in Barrett's esophagus because treatment with the COX-2 inhibitor hinders proliferation of Barrett's Esophageal epithelial cells in culture, but proliferation is restored by treatment with prostaglandin.
136
COX-2 induction by unconjugated bile acids involves reactive oxygen species-mediated signalling pathways in Barrett’s oesophagus and oesophageal adenocarcinoma
TL;DR: Unconjugated bile acids induce CREB and AP-1-dependent COX-2 expression in Barrett’s oesophagus and OA through ROS-mediated activation of PI3K/AKT and ERK1/2.