Muye He
4 Papers
3 Citations
Muye He is an academic researcher. The author has contributed to research in topics: Gene silencing & Dopaminergic. The author has an hindex of 1, co-authored 1 publications.
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Papers
Neuronal actin cytoskeleton gain of function in the human brain
Kinga Szigeti,I. Ihnatovych,Nicolás Matías Rosas,R. Dorn,Emily Notari,Eduardo Cortes Gomez,Muye He,Ivan V. Maly,Shreyas Prasad,Erik Nimmer,Yun Heo,Beata Fuchsova,David A. Bennett,Wilma A. Hofmann,Arnd Pralle,Yongho Bae,Jianmin Wang +16 more
TL;DR: CHRFAM7A may facilitate neuronal adaptation to changes in the brain environment in physiological and pathological conditions contributing to risk or recovery and requires human studies in the areas of memory formation and erasure, cognitive reserve, and neuronal plasticity.
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Deep Brain Magnetothermal Silencing of Dopaminergic Neurons via Endogenous TREK1 Channels Abolishes Place Preference in Mice
TL;DR: TREK1 is a highly efficient, thermally activated neuronal silencer and magnetothermal neuronal silencing using endogenous TREK-1 channels can suppress the dopaminergic reward in the ventral tegmental area and the place preference in mice.
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Spatially resolving membrane tension via fluorescence single-channel flux recording in Piezo1 channels
Muye He,Yue Xing,Michael Zucker,Philip A. Gottlieb,Arnd Pralle +4 more
TL;DR: Researchers developed a Piezo1 channel that optically reports local membrane tension at the single molecule level, enabling spatial resolution of membrane tension and forces across the cell surface, shedding light on cell actions and tissue homeostasis.
CHRFAM7A diversifies human immune adaption through Ca2+ signalling and actin cytoskeleton reorganization.
Kinga Szigeti,I. Ihnatovych,Emily Notari,Ryu P Dorn,Ivan Maly,Muye He,Barbara Birkaya,Shreyas Prasad,Robin Schwartz Byrne,Dinesh C Indurthi,Erik Nimmer,Yuna Heo,Kolos Retfalvi,Lee Chaves,Norbert Sule,Wilma A Hofmann,Anthony Auerbach,Gregory Wilding,Yongho Bae,Jessica Reynolds +19 more
TL;DR: CHRFAM7A introduces an additional, human specific, layer to Ca2+ regulation leading to an innate immune gain of function through the actin cytoskeleton it drives adaptation to the mechanical properties of the tissue environment leading to an ability to invade previously immune restricted niches.