Minna Anthoni
Finnish Institute of Occupational Health
6 Papers
51 Citations
Minna Anthoni is an academic researcher from Finnish Institute of Occupational Health. The author has contributed to research in topics: Chemokine & Proinflammatory cytokine. The author has an hindex of 5, co-authored 6 publications. Previous affiliations of Minna Anthoni include University of Helsinki.
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Papers
Transforming growth factor-beta/Smad3 signalling regulates inflammatory responses in a murine model of contact hypersensitivity.
TL;DR: This work has shown that the Smad proteins, which are intracellular TGF‐β signal transducers, mediate most actions of T GF‐β.
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Smad3 signal transducer regulates skin inflammation and specific IgE response in murine model of atopic dermatitis.
Minna Anthoni,Guoying Wang,Guoying Wang,Chu-Xia Deng,Henrik Wolff,Henrik Wolff,Antti Lauerma,Harri Alenius +7 more
TL;DR: It is demonstrated that the Smad3-pathway regulates allergen-induced skin inflammation and systemic IgE antibody production in a murine model AD and might be a potential target in the therapy of allergic skin diseases.
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Smad3 Regulates Dermal Cytokine and Chemokine Expression and Specific Antibody Production in Murine Responses to a Respiratory Chemical Sensitizer
TL;DR: The Smad3 molecule contributes significantly to the regulation of the cytokine and chemokine network during the contact hypersensitivity (CHS) response to TMA.
6
Invariant Natural Killer T Cells Play a Role in Chemotaxis, Complement Activation and Mucus Production in a Mouse Model of Airway Hyperreactivity and Inflammation.
Piia Karisola,Maili Lehto,Pia Anneli Sofia Kinaret,Niina Ahonen,Rita Haapakoski,Minna Anthoni,Masaru Taniguchi,Henrik Wolff,Anne Puustinen,Harri Alenius +9 more
TL;DR: Differentially abundant proteins in the BALF proteome of α-GalCer-treated wild type mice included lungkine, pulmonary surfactant-associated protein D (SFTPD), calcium-activated chloride channel regulator 1 (CLCA1), fragments of complement 3, chitinase 3-like proteins 1 and 3 (CH3LI and 3) and neutrophil gelatinase-associated lipocalin (NGAL).
Chemokine responses distinguish chemical-induced allergic from irritant skin inflammation: Memory T cells make the difference
Stephan Meller,Antti Lauerma,Frank Kopp,Franziska Winterberg,Minna Anthoni,Anja Müller,Michael Gombert,Anna Haahtela,Harri Alenius,Juliane Rieker,Marie-Caroline Dieu-Nosjean,Robert Kubitza,Ernst Gleichmann,Thomas Ruzicka,Albert Zlotnik,Bernhard Homey +15 more
TL;DR: Important insights are provided into the molecular basis of chemical-induced allergic and irritant contact dermatitis, identify novel markers suitable for their differentiation, and demonstrate the cooperation of inflammatory and homeostatic chemokines in the recruitment of pathogenic leukocyte subsets.