Mingming Lei
China Medical University (PRC)
9 Papers
49 Citations
Mingming Lei is an academic researcher from China Medical University (PRC). The author has contributed to research in topics: Acute coronary syndrome & Myocardial infarction. The author has an hindex of 5, co-authored 9 publications.
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Papers
A brief review: the therapeutic potential of bone marrow mesenchymal stem cells in myocardial infarction.
TL;DR: The critical issue the authors must now address is the reduced viability of transplanted BMSCs due to inadequate blood supply, poor nourishment of cells, and generation of free radicals.
ALDH2 rs671 Polymorphism and Coronary Heart Disease Risk Among Asian Populations: A Meta-Analysis and Meta-Regression
TL;DR: The current meta-analysis indicates that the A variant of the ALDH2 rs671 polymorphism may increase the risk of both CHD and MI among Asian populations.
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Seven functional polymorphisms in the CETP gene and myocardial infarction risk: a meta-analysis and meta-regression.
TL;DR: It is suggested that CETP rs708272 (C>T) and rs1800775 (C>.A) polymorphisms may contribute to MI susceptibility, especially among Caucasians, and may be promising and potential biomarkers for early diagnosis of MI.
Associations between circulating IgG antibodies to Apolipoprotein B100-derived peptide antigens and acute coronary syndrome in a Chinese Han population.
TL;DR: Circulating IgG for ApoB100-derived peptide antigens may be a useful biomarker of ACS, although anti-ApoB IgG levels were not associated with the coronary artery plaque burden characterized by the coronary Gensini score.
MiR-17-5p impairs trafficking of H-ERG K+ channel protein by targeting multiple er stress-related chaperones during chronic oxidative stress.
TL;DR: Deregulation of the miR-17-5p seed family miRNAs can cause severe impairment of h-ERG trafficking through targeting multiple ER stress-related chaperones, and activation of AP1 likely accounts for the deleterious upregulation of these mi RNAs, in the setting of prolonged duration of oxidative stress.