10 Papers
5 Citations
Ming Ni is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Medicine & Inflammation. The author has an hindex of 3, co-authored 5 publications. Previous affiliations of Ming Ni include Nanjing Medical University.
Chat about Author
Papers
FSTL1 promotes liver fibrosis by reprogramming macrophage function through modulating the intracellular function of PKM2
Jianhua Rao,Hao Wang,Ming Ni,Zeng Wang,Zhiyi Wang,Song Wei,Mu Liu,Peng Wang,Jiannan Qiu,Lei Zhang,Chen Wu,Hongbing Shen,Xuehao Wang,Feng Cheng,Ling Lu +14 more
TL;DR: Macrophage FSTL1 promotes the progression of liver fibrosis by inducing M1 polarisation and inflammation based on the intracellular PKM2 reprogramming function of macrophages.
T-Cell Immunoglobulin and Mucin Domain-Containing Protein-4 Is Critical for Kupffer Cell Homeostatic Function in the Activation and Resolution of Liver Ischemia Reperfusion Injury
Ming Ni,Ming Ni,Jing Zhang,Rebecca A. Sosa,Hanwen Zhang,Han Wang,Dan Jin,Kaitlyn Crowley,Bita V. Naini,F Elaine Reed,Ronald W. Busuttil,Jerzy W. Kupiec-Weglinski,Xuehao Wang,Yuan Zhai +13 more
TL;DR: In this paper, the authors dissected roles of liver resident macrophage Kupffer cells (KCs), with a functional focus on efferocytosis receptor T-cell immunoglobulin and mucin domain-containing protein-4 (TIM-4), in both the activation and resolution of IRI in a murine liver partial warm ischemia model.
49
Targeting Notch1-YAP Circuit Reprograms Macrophage Polarization and Alleviates Acute Liver Injury in Mice
TL;DR: In this paper , the Notch and Hippo-YAP pathway has been investigated for controlling macrophage polarization in acute liver injury, raising the possibility of targeting macrophages Notch1-yAP circuit as an effective strategy for liver inflammation-related diseases.
19
Gsk3β regulates the resolution of liver ischemia/reperfusion injury via MerTK
Han-Ming Zhang,Ming Ni,Han Wang,Jing Zhang,Dan Jin,Ronald W. Busuttil,Jerzy W. Kupiec-Weglinski,Wei Li,Xuehao Wang,Yuan Zhai +9 more
TL;DR: In this paper , the authors found that Gsk3β inhibitory phosphorylation increased at both the early-activation and late-resolution stages of the liver IRI in both WT and GSK3β-KO mice.
Isoform- and Cell Type-Specific Roles of Glycogen Synthase Kinase 3 N-Terminal Serine Phosphorylation in Liver Ischemia Reperfusion Injury.
Ming Ni,Haoming Zhou,Haoming Zhou,Jing Zhang,Dan Jin,Dan Jin,Tianfei Lu,Tianfei Lu,Ronald W. Busuttil,Jerzy W. Kupiec-Weglinski,Xuehao Wang,Yuan Zhai +11 more
TL;DR: Gsk3 N-terminal serine phosphorylation inhibits liver innate immune activation but suppresses hepatocyte autophagy in response to inflammation.
5