Merryman Wd
Vanderbilt University
4 Papers
9 Citations
Merryman Wd is an academic researcher from Vanderbilt University. The author has contributed to research in topics: Macrophage & Cell. The author has an hindex of 1, co-authored 4 publications.
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Papers
Cell Programmed Nutrient Partitioning in the Tumor Microenvironment
Bradley I. Reinfeld,Bradley I. Reinfeld,Matthew Z. Madden,Matthew Z. Madden,Melissa M. Wolf,Melissa M. Wolf,Anna Chytil,Jackie E. Bader,AR Patterson,Allison S. Cohen,Ahmed Ali,Ahmed Ali,Brian T. Do,Brian T. Do,Alexander Muir,Caroline A. Lewis,Rachel Hongo,Kirsten Young,Rachel E. Brown,Rachel E. Brown,Vera M. Todd,Vera M. Todd,Tessa Huffstater,Abin Abraham,Abin Abraham,Richard T. O’Neil,Richard T. O’Neil,Matthew H. Wilson,Matthew H. Wilson,F Xin,Mohammed N. Tantawy,Merryman Wd,Rachelle W. Johnson,Christopher S. Williams,Christopher S. Williams,Christopher S. Williams,Emily F. Mason,Frank M. Mason,Katy Beckermann,M. G. Vander Heiden,M. G. Vander Heiden,HC Manning,Jeffrey C. Rathmell,Wendy Kimryn Rathmell +43 more
TL;DR: Cell selective partitioning of glucose and glutamine can be exploited to develop therapies and imaging strategies to alter the metabolic programs of specific cell populations in the TME, and it is observed that intrinsic cellular programs can play a major role in the use of some nutrients.
Radiofrequency Ablation Alters the Microstructural Organization of Healthy and Enzymatically Digested Porcine Mitral Valves
TL;DR: Investigation of mechanisms by which radiofrequency ablation alters the geometry, microstructural organization, and mechanical properties of healthy and digested leaflets found this technique may be a therapeutic approach for myxomatous mitral valve disease.
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Focal adhesion kinase regulates early steps of myofibrillogenesis in cardiomyocytes
TL;DR: A fundamental mechanism regulating the maturation of myofibrils in human cardiomyocytes is uncovered, and a key role for Focal adhesion kinase (FAK), a known regulator of adhesion dynamics in non-muscle cells, is established in regulating focal adhesion Dynamics in cardiomeocytes.
Macrophages Promote Aortic Valve Cell Calcification Through STAT3 Splicing
Michael A. Raddatz,Tessa Huffstater,Matthew R. Bersi,Bradley I. Reinfeld,Matthew Z. Madden,Sabrina E. Booton,Rathmell Wk,Jeffrey C. Rathmell,Brian R. Lindman,Meena S. Madhur,Merryman Wd +10 more
TL;DR: This study reveals that Notch1+/- aortic valve disease involves increased macrophage recruitment and polarization driven by altered aortsic valve cell secretion, and that increased macophage recruitment promotes osteogenic calcification through STAT3 splicing changes.