Meeta Gera
Jeju National University
21 Papers
46 Citations
Meeta Gera is an academic researcher from Jeju National University. The author has contributed to research in topics: Medicine & Metastasis. The author has an hindex of 9, co-authored 18 publications. Previous affiliations of Meeta Gera include Amity University.
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Papers
Nanoformulations of curcumin: an emerging paradigm for improved remedial application.
Meeta Gera,Neelesh Sharma,Mrinmoy Ghosh,Do Luong Huynh,Sung-Jin Lee,Taesun Min,Taeho Kwon,Dong Kee Jeong +7 more
TL;DR: Recent nanotechnology based implementations applied for overcoming the innate constraints of native curcumin and additionally the associated challenges which restrict its potential therapeutic applications both in vivo and in-vitro studies, as well as their detailed mechanism of action have additionally been discussed.
Antifungals and Drug Resistance
Chowdhury Mobaswar Hossain,Lisa K. Ryan,Meeta Gera,Sabyasachi Choudhuri,Nazmun Lyle,Kazi Asraf Ali,Gill Diamond +6 more
TL;DR: Bacteria have an amazing capacity to become resistant to antibiotic action as well, and the effectiveness of the scarce antifungal arsenal is jeopardised by this antibiotic resistance, which poses a severe threat to public health.
Transformation of animal genomics by next-generation sequencing technologies: a decade of challenges and their impact on genetic architecture
TL;DR: The current state of sequencing methods is explored, many of which are already used in animal genomic studies, and the state of cattle, swine, horse, and chicken genome sequencing is summarized and its achievements during the last few years are illustrated.
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Wogonin suppresses stem cell-like traits of CD133 positive osteosarcoma cell via inhibiting matrix metallopeptidase-9 expression.
Do Luong Huynh,Taeho Kwon,Jiao Jiao Zhang,Neelesh Sharma,Meeta Gera,Mrinmoy Ghosh,Nameun Kim,Somi Kim Cho,Dong-Sun Lee,Yang Ho Park,Dong Kee Jeong +10 more
TL;DR: By inhibiting the formation of and reducing the size of spheres, Wogonin at a concentration of 40–80 μM effectively minimizes potential risk from CSC and demonstrates a new approach for developing a potential therapy for osteosarcoma.
BRM270 inhibits cancer stem cell maintenance via microRNA regulation in chemoresistant A549 lung adenocarcinoma cells.
Taeho Kwon,Nisansala Chandimali,Do Luong Huynh,Jiao Jiao Zhang,Nameun Kim,Yesol Bak,Do-Young Yoon,Dae-Yeul Yu,Jae Cheol Lee,Meeta Gera,Mrinmoy Ghosh,Yang Ho Park,Dong Kee Jeong +12 more
TL;DR: It is reported that the growth, migration, and invasion of normal human lung adenocarcinoma cells and their chemoresistant derivatives was inhibited by BRM270 treatment, andBRM270 was found to modulate CSC self-renewal and tumor-initiating capacity via positive regulation of the miRNA-128.