Mats Sandberg
Sahlgrenska University Hospital
6 Papers
198 Citations
Mats Sandberg is an academic researcher from Sahlgrenska University Hospital. The author has contributed to research in topics: Extracellular & Taurine. The author has an hindex of 6, co-authored 6 publications.
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Papers
Cellular origins of endogenous amino acids released into the extracellular fluid of the rat striatum during severe insulin-induced hypoglycemia.
TL;DR: Data suggest that GABA, Glu, and Asp are released primarily from their transmitter pools during severe hypoglycemia, and the releasable pools of Tau and PEA appear to be located in kainate‐sensitive striatal neurons.
138
Electroporation of single cells and tissues with an electrolyte-filled capillary.
Kerstin Nolkrantz,Cecilia Farre,Anke Brederlau,Roger Karlsson,Carrie Brennan,Peter S. Eriksson,Stephen G. Weber,Mats Sandberg,Owe Orwar +8 more
TL;DR: It is shown how an electrolyte-filled capillary coupled to a high-voltage power supply can be used as a versatile electroporation tool for the delivery of dyes, drugs, and biomolecules to the cytoplasm of single cells and cells in tissues.
114
Changes in extracellular amino acids and spontaneous neuronal activity during ischemia and extended reflow in the CA1 of the rat hippocampus.
TL;DR: The postischemic increase of excitatory amino acids and the recovery of the neuronal activity may stress the CA1 pyramidal cells, which could be detrimental in combination with, e.g., postsynaptic impairments.
107
Release of endogenous amino acids from superior colliculus of the rabbit: in vitro studies after retinal ablation.
Mats Sandberg,Lanfranco Corazzi +1 more
TL;DR: The results do not favour glutamate as the major optic nerve transmitter in the rabbit, but do not rule out glutamate as a transmitter in a minor population of retinal fibres.
47
Effect of serine and ethanolamine administration on phospholipid-related compounds and neurotransmitter amino acids in the rabbit hippocampus.
TL;DR: Mechanisms for the increase of extracellular levels of ethanolamine and phosphoethanolamine in CNS regions, such as the hippocampus, in transient brain ischemia, hypoglycemia, seizures, etc, are concerned.
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