Mathieu Jablonski
Paris Diderot University
6 Papers
19 Citations
Mathieu Jablonski is an academic researcher from Paris Diderot University. The author has contributed to research in topics: Autoantibody & Immunoglobulin E. The author has an hindex of 5, co-authored 6 publications.
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Papers
Autoreactive IgE Is Prevalent in Systemic Lupus Erythematosus and Is Associated with Increased Disease Activity and Nephritis
Barbara Dema,Christophe Pellefigues,Sarfaraz Hasni,Nathalie Gault,Chao Jiang,Tiffany K. Ricks,Michael Bonelli,Jörg Scheffel,Karim Sacre,Mathieu Jablonski,Delphine Gobert,Thomas Papo,Eric Daugas,Gabor G. Illei,Nicolas Charles,Juan Rivera +15 more
TL;DR: It is argued that the overall levels of IgE autoantibodies, independently or in combination with IgG autoantigens, may serve as indicators of active disease.
Immunoglobulin E plays an immunoregulatory role in lupus.
Barbara Dema,Barbara Dema,Nicolas Charles,Christophe Pellefigues,Tiffany K. Ricks,Ryo Suzuki,Chao Jiang,Jörg Scheffel,Sarfaraz Hasni,Victoria Hoffman,Mathieu Jablonski,Karim Sacre,Delphine Gobert,Thomas Papo,Eric Daugas,Steve P. Crampton,Silvia Bolland,Juan Rivera +17 more
TL;DR: It is shown that deficiency of IgE delays lupuslike disease development and severity, as demonstrated by reduced autoantibody production and amelioration of organ pathologies.
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Prostaglandin D 2 amplifies lupus disease through basophil accumulation in lymphoid organs
Christophe Pellefigues,Barbara Dema,Yasmine Lamri,Fanny Saidoune,Nathalie Chavarot,Charlotte Lohéac,Emeline Pacreau,Michael Dussiot,Caroline Bidault,Florian Marquet,Mathieu Jablonski,Jonathan M. Chemouny,Fanny Jouan,Antoine Dossier,Marie-Paule Chauveheid,Delphine Gobert,Thomas Papo,Hajime Karasuyama,Karim Sacre,Eric Daugas,Nicolas Charles +20 more
TL;DR: A role for prostaglandin D2 (PGD2) is shown in the pathophysiology of SLE, where accumulation is driven by PGD2-induced CXCR4 surface expression and trafficking of basophils, which affects pathogenesis by stimulating autoantibody production.
Necrotizing fasciitis during de novo minimal change nephrotic syndrome in a kidney transplant recipient.
Vincent Audard,Agathe Pardon,O. Claude,Mathieu Jablonski,P. Remy,Dominique Desvaux,Laurent Lantieri,Peter Lang,Philippe Grimbert +8 more
TL;DR: The development of de novo minimal‐change nephrotic syndrome after renal allograft transplantation should alert physicians to the possibility of MRSA NF during an increase in the immunosuppressive regimen.
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