Masayoshi Abe
Fukuoka University
26 Papers
240 Citations
Masayoshi Abe is an academic researcher from Fukuoka University. The author has contributed to research in topics: Anaphylatoxin & Allergic inflammation. The author has an hindex of 12, co-authored 26 publications.
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Papers
Evaluating the role of inducible nitric oxide synthase using a novel and selective inducible nitric oxide synthase inhibitor in septic lung injury produced by cecal ligation and puncture
Iku Okamoto,Masayoshi Abe,Kazuhiko Shibata,Naomi Shimizu,Noriyuki Sakata,Takeshi Katsuragi,Keiichi Tanaka +6 more
TL;DR: Intervention with a potent and selective iNOS inhibitor seemed to improve survival in CLP rats when used at the appropriate doses and time points, however, the self-limited mechanism of iN OS regulation in the lungs may also indicate that iNos plays only a limited role in sepsis and septic shock.
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Contribution of anaphylatoxin C5a to late airway responses after repeated exposure of antigen to allergic rats.
Masayoshi Abe,Kazuhiko Shibata,Hiroyasu Akatsu,Naomi Shimizu,Noriyuki Sakata,Takeshi Katsuragi,Hidechika Okada +6 more
TL;DR: The contribution of complement to allergic asthma was elucidated and up-regulation of C5aR mRNA was shown in lungs after triple OVA exposure, but almost no up- regulation of C3aR.
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Effects of inducible nitric oxide synthase inhibitors on asthma depending on administration schedule.
Masayoshi Abe,Yuri Hayashi,Akira Murai,Kazuhiko Shibata,Noriyuki Sakata,Rie Igarashi,Takeshi Katsuragi,Keiichi Tanaka +7 more
TL;DR: Although iNOS inhibitors may be beneficial for asthma, repeated administration may be detrimental because of extensive reduction of NO and downregulation of SOD.
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Intratracheal administration of anaphylatoxin C5a potentiates antigen-induced pulmonary reactions through the prolonged production of cysteinyl-leukotrienes.
Mamoru Kodani,Noriyuki Sakata,Yukio Takano,Hiro-o Kamiya,Takeshi Katsuragi,Tony E. Hugli,Masayoshi Abe +6 more
TL;DR: It is suggested that anaphylatoxin C5a may mediate the airway inflammatory response induced by a specific antigen challenge partly through a prolonged production of cysLTs and the release of histamine.
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Distribution of Rat C5a Anaphylatoxin Receptor
Hiroyasu Akatsu,Masayoshi Abe,Takashi Miwa,Hisashi Tateyama,Seiji Maeda,Noriko Okada,Kiyohide Kojima,Hidechika Okada +7 more
TL;DR: C5aR was usually expressed at the site of exposure to pathogens, such as in salivary gland or lung, and was up‐regulated in liver in the inflammatory state induced by lipopolysaccharide (LPS) administration and was not accompanied by an increase in C 5aR mRNA in Kupffer cells following LPS challenge.
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