Masato Kakeda
Mie University
30 Papers
117 Citations
Masato Kakeda is an academic researcher from Mie University. The author has contributed to research in topics: Medicine & Antigen. The author has an hindex of 15, co-authored 30 publications. Previous affiliations of Masato Kakeda include University of Bern.
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Papers
Granzyme B is a novel interleukin-18 converting enzyme.
Youichi Omoto,Keiichi Yamanaka,Kazuya Tokime,Shigehisa Kitano,Masato Kakeda,Tomoko Akeda,Ichiro Kurokawa,Esteban C. Gabazza,Hiroko Tsutsui,Naoyuki Katayama,Kiyofumi Yamanishi,Kenji Nakanishi,Hitoshi Mizutani +12 more
TL;DR: GrB is a potent IL-18 converting enzyme and suggest that GrB secreted by CTLs and/or NK cells may initiate IL- 18 release from target cells, leading to the development of inflammation.
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Cytokines and chemokines in irritant contact dermatitis.
TL;DR: This review will focus on key cytokines and chemokines involved in the pathogenesis of irritant contact dermatitis and also contrast the differences between allergic contact dermatitus and irritantContact dermatitis.
Calcineurin Inhibitors Suppress Cytokine Production from Memory T Cells and Differentiation of Naïve T Cells into Cytokine-Producing Mature T Cells
Kenshiro Tsuda,Keiichi Yamanaka,Hiroshi Kitagawa,Tomoko Akeda,Masanao Naka,Kaori Niwa,Takehisa Nakanishi,Masato Kakeda,Esteban C. Gabazza,Hitoshi Mizutani +9 more
TL;DR: The present study focused on the effects of CsA and Tac on cytokine secretion from purified human memory CD4(+)T cells and the differentiation of naïve T cells into cytokine-producing memory T cells.
Increased interferon-γ, interleukin-12p40 and IL-8 production in Propionibacterium acnes-treated peripheral blood mononuclear cells from patient with acne vulgaris: Host response but not bacterial species is the determinant factor of the disease
Hitomi Sugisaki,Keiichi Yamanaka,Masato Kakeda,Hiroshi Kitagawa,Kaori Tanaka,Kunitomo Watanabe,Esteban C. Gabazza,Ichiro Kurokawa,Hitoshi Mizutani +8 more
TL;DR: The inflammatory response of acne appears to be attributable to P. acnes-induced host immune response rather than P.Acne vulgaris, and the bacteriological factor in the pathogenesis of acne is evaluated.
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