Martin Hindermann
Max Planck Society
5 Papers
12 Citations
Martin Hindermann is an academic researcher from Max Planck Society. The author has contributed to research in topics: Hippocampal formation & Hippocampus. The author has an hindex of 2, co-authored 5 publications.
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Papers
Hippocampal neurons respond to brain activity with functional hypoxia.
Umer Javed Butt,Agnes A. Steixner-Kumar,Constanze Depp,Ting Sun,Ting Sun,Imam Hassouna,Liane Wüstefeld,Sahab Arinrad,Matthias R. Zillmann,Nadine Schopf,Laura Fernandez Garcia-Agudo,Leonie Mohrmann,Ulli Bode,Anja Ronnenberg,Martin Hindermann,Sandra Goebbels,Stefan Bonn,Dörthe M. Katschinski,Kamilla W. Miskowiak,Klaus-Armin Nave,Hannelore Ehrenreich +20 more
TL;DR: In this article, a refined transgenic approach in mice, utilizing the oxygen-dependent degradation (ODD) domain of HIF-1α fused to CreERT2 recombinase, allows them to demonstrate hypoxic cells in the performing brain under normoxia and motor-cognitive challenge, and spatially map them by light-sheet microscopy, all in comparison to inspiratory hypoxia as strong positive control.
Evaluation of a mechanical lung model to test small animal whole body plethysmography.
TL;DR: In this paper, an artificial lung model was designed to evaluate different predictions about and around whole-body plethysmography (WBP) in animals and animal models of a variety of diseases.
Autoantibodies against NMDA receptor 1 modify rather than cause encephalitis.
Justus B H Wilke,Martin Hindermann,Stefan A. Berghoff,Svenja Zihsler,Sahab Arinrad,Anja Ronnenberg,Nadine Barnkothe,Agnes A. Steixner-Kumar,Stefan Röglin,Winfried Stöcker,Michael Hollmann,Klaus-Armin Nave,Fred Lühder,Hannelore Ehrenreich +13 more
TL;DR: In this paper, the etiology and pathogenesis of "anti-N-methyl-D-aspartate-receptor (NMDAR) encephalitis" and the role of autoantibodies (AB) in this condition are still obscure.
Whole-body plethysmography revisited
TL;DR: In this article, a simple device was designed to serve as an artificial lung model that enables a thorough evaluation of different predictions about and around whole-body plethysmography.
Inducing sterile pyramidal neuronal death in mice to model distinct aspects of gray matter encephalitis.
Justus B H Wilke,Martin Hindermann,Amir Moussavi,Umer Javed Butt,Rakshit Dadarwal,Rakshit Dadarwal,Stefan A. Berghoff,Aref Kalantari Sarcheshmeh,Anja Ronnenberg,Svenja Zihsler,Sahab Arinrad,Rüdiger Hardeland,Jan Seidel,Fred Lühder,Klaus-Armin Nave,Susann Boretius,Susann Boretius,Hannelore Ehrenreich +17 more
TL;DR: In this article, the authors used adult male NexCreERT2xRosa26-eGFP-DTA mice for the induction of a sterile encephalitis by diphtheria toxin-mediated ablation of cortical and hippocampal pyramidal neurons which also recruits immune cells into gray matter.