Martin Berry
University of Birmingham
18 Papers
160 Citations
Martin Berry is an academic researcher from University of Birmingham. The author has contributed to research in topics: Axon & Neurite. The author has an hindex of 12, co-authored 18 publications. Previous affiliations of Martin Berry include King's College London.
Chat about Author
Papers
Caspases in retinal ganglion cell death and axon regeneration.
TL;DR: The molecular signalling mechanisms of apoptotic and inflammatory caspase responses in R GC specifically, their involvement in RGC degeneration and their potential as therapeutic targets are discussed.
Patent
Devices containing DNA encoding neurotrophic agents and related compositions and methods
Andrew Baird,Ana Maria Gonzalez,Ann Logan,Martin Berry +3 more
- 17 Jan 2003
TL;DR: In this paper, the authors describe devices useful in the delivery of DNA encoding neurotrophic agents, anti-fibrotic agents, and related compositions for use in the treatment of central and/or peripheral nervous system injury.
61
Prospects for mTOR-mediated functional repair after central nervous system trauma.
TL;DR: The therapeutic potential of a mTOR-based treatment aimed at promoting functional recovery in CNS trauma patients is discussed, recognising that to fulfil this ambition, the new therapy should aim to promote not only axon regeneration but also remyelination of regenerated axons, neuronal survival and re-innervation of denervated targets through accurate axonal guidance and synaptogenesis, all with minimal adverse effects.
58
Patent
Compositions and methods for delivery of agents for neuronal regeneration and survival
Andrew Baird,Ana Maria Gonzalez,Ann Logan,Martin Berry +3 more
- 23 Oct 1998
TL;DR: In this paper, the authors describe devices useful in the delivery of DNA encoding neurotrophic agents, anti-fibrotic agents, and related compositions for use in the treatment of central and/or peripheral nervous system injury.
44
Epidermal growth factor receptor inhibitors promote CNS axon growth through off-target effects on glia.
TL;DR: It is suggested that chemical EGFR inhibitors act independently of EGFR, inducing glia and neurons to secrete NT and raising cAMP levels in DRG cultures, leading to Trk-dependent disinhibited DRGN neurite outgrowth.
39