Mark Perkins
University of Wisconsin-Madison
15 Papers
217 Citations
Mark Perkins is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Medial forebrain bundle & Hippocampal formation. The author has an hindex of 11, co-authored 12 publications. Previous affiliations of Mark Perkins include University of Wisconsin–Whitewater.
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Papers
Muscarinic blockade weakens interaction of gamma with theta rhythms in mouse hippocampus.
TL;DR: Investigation of the effects of a blockade of muscarinic acetylcholine receptors by atropine on θ and γ oscillations, and their interaction, in mouse hippocampus in vivo demonstrates that phase locking of the amplitude of θ oscillations to θ in hippocampal area CA1 is partially governed by neuronal elements harbouring mus carinic receptors.
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Slowing of the hippocampal θ rhythm correlates with anesthetic-induced amnesia
Misha Perouansky,Vinuta Rau,Tim Ford,S. Irene Oh,Mark Perkins,Edmond I. Eger,Robert A. Pearce +6 more
TL;DR: At subhypnotic amnestic concentrations, &thGr;-oscillation frequency was the parameter most consistently affected by these three anesthetics, consistent with the hypothesis that modulation of the &thgr; rhythm contributes to anesthetic-induced amnesia.
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Altered GABAA,slow Inhibition and Network Oscillations in Mice Lacking the GABAA Receptor β3 Subunit
Harald Hentschke,Claudia Benkwitz,Matthew I. Banks,Mark Perkins,Gregg E. Homanics,Robert A. Pearce +5 more
TL;DR: A strong contribution of beta(3) subunits to slow GABAergic inhibition onto pyramidal neurons but not onto GABA(A,fast) -producing interneurons is pointed to and different roles for these slow inhibitory synapses in the generation and coordination of hippocampal network rhythms are supported.
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Temporary inactivation of the retrorubral fields decreases the rewarding effect of medial forebrain bundle stimulation
Meg Waraczynski,Mark Perkins +1 more
TL;DR: Results indicate that some neural elements located in the RRF contribute to the rewarding effect of MFB stimulation, and possible roles for these elements in the anatomical substrate for MFB self-stimulation are discussed.
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Lesions of pontomesencephalic cholinergic nuclei do not substantially disrupt the reward value of medial forebrain bundle stimulation
Meg Waraczynski,Mark Perkins +1 more
TL;DR: The results are interpreted in terms of a previously proposed hypothesis regarding the role in MFB self-stimulation of ascending cholinergic input from the pontomesencephalon to ventral tegmental dopaminergic neurons.
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