Mark A. Miller
West Virginia University
8 Papers
40 Citations
Mark A. Miller is an academic researcher from West Virginia University. The author has contributed to research in topics: Dementia & Stimulus control. The author has an hindex of 5, co-authored 6 publications.
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Papers
Cocaine's effects on food-reinforced pecking in pigeons depend on food-deprivation level.
TL;DR: Low doses of cocaine increased low baseline rates of pecking in the initial portions of the fixed-interval schedules by a greater magnitude when pigeons were more food deprived, and altered both the rate-decreasing and rate-increasing effects of cocaine.
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Drug discrimination using a Pavlovian conditional discrimination paradigm in pigeons.
TL;DR: Three pigeons were studied using a discriminated autoshaping procedure in which the presence or absence of methadone served as a conditional stimulus signalling which of two key light CSs would be followed by grain access.
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Frontal and temporal lobe correlates of verbal learning and memory in aMCI and suspected Alzheimer's disease dementia.
Cierra M. Keith,Marc W. Haut,K. Wilhelmsen,Rashi I. Mehta,Mark A. Miller,R. Navia,Melanie Ward,K. Lindberg,Michelle Coleman,William T McCuddy,Gerard Deib,Angelo Clark Giolzetti,Pierre-François D'Haese +12 more
TL;DR: This paper examined the role of frontal lobes in learning, recognition, and retention of new verbal information, as well as the presence of specific errors (i.e., intrusions and false-positive errors).
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Searching for Evidence of Transfer between Drug Facilitators
TL;DR: The lack of transfer of modulatory control by drug stimuli in Experiments 1–3, coupled with its occurrence in Experiment 4 and previous studies, suggests that stimulus control by drugs may differ in important ways from stimulus Control by conventional stimuli.
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Discrimination of methadone and cocaine by pigeons without explicit discrimination training
TL;DR: The results suggest that discriminative control by methadone and cocaine was established without explicit discrimination training and steepened dose functions for the training drugs, and the effects of several other substituted drugs depended on the pharmacology of the training drug.
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