Mariola Andrejko
Maria Curie-Skłodowska University
25 Papers
100 Citations
Mariola Andrejko is an academic researcher from Maria Curie-Skłodowska University. The author has contributed to research in topics: Galleria mellonella & Hemolymph. The author has an hindex of 8, co-authored 21 publications.
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Papers
Diverse effects of Galleria mellonella infection with entomopathogenic and clinical strains of Pseudomonas aeruginosa.
TL;DR: In this in vivo study on the G. mellonella–P.
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Three Pseudomonas aeruginosa strains with different protease profiles.
TL;DR: The proteolytic activity of three Pseudomonas aeruginosa strains, ATCC 27853 - a reference strain, and two clinical isolates was tested and confirmed the presence of both the lasB gene encoding elastase B and aprA coding for alkaline protease in the genomes of the three P. aerug inosa strains analyzed.
Antibacterial activity in vivo and in vitro in the hemolymph of Galleria mellonella infected with Pseudomonas aeruginosa.
TL;DR: The antibacterial activity of hemolymph from Galleria mellonella infected with entomopathogenic strain of Pseudomonas aeruginosa and non-pathogenic bacterium Escherichia coli was studied and it was shown that elastase B degraded synthetic cecropin B while in the presence of 6 mM EDTA antib bacterial activity of cell-free hemoly mph was not changed which confirmed that the activity was abolished by the metalloprotease.
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Galleria mellonella hemocytes destruction after infection with Pseudomonas aeruginosa.
TL;DR: The influence of infection with an entomopathogenic strain of Pseudomonas aeruginosa on Galleria mellonella hemocytes was investigated and extensive bacteriaemia developed 18 h after infection, indicating that hemocytes become dead ultimately.
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Changes in Galleria mellonella apolipophorin III level during Pseudomonas aeruginosa infection.
TL;DR: The results of studies suggest that a significant part of total proteolytical activity in the plasma of infected G. mellonella larvae can be attributed to proteases produced by P. aeruginosa during pathogenesis.
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