Marie D. Sauro
Syracuse University
16 Papers
89 Citations
Marie D. Sauro is an academic researcher from Syracuse University. The author has contributed to research in topics: Protein kinase C & Blood pressure. The author has an hindex of 10, co-authored 16 publications. Previous affiliations of Marie D. Sauro include University of Massachusetts Dartmouth.
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Papers
A study of polypharmacy with second generation antipsychotics in patients with severe and persistent mental illness.
TL;DR: Polypharmacy with SGAs is quite frequent among chronic inpatients with severe and persistent mental illness despite a limited empirical database supporting its use, and the results of this pilot study do not demonstrate the effectiveness and safety of this practice.
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Gabapentin's Effect on Agitation in Severely and Persistently Mentally III Patients
TL;DR: Adjunctive gabapentin appears to be associated with a reduction in agitation in chronically hospitalized SPMI patients, and controlled, prospective trials are needed before any definitive conclusion can be drawn regarding the role of gabAPentin in the treatment of this group of patients.
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The anti-tumor agent, taxol, attenuates contractile activity in rat aortic smooth muscle.
TL;DR: The data suggest that taxol does not augment hypersensitivity of the vascular, but instead attenuates contractile activity and may have important implications with respect to treatment of women with both cancer and cardiovascular disease.
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Seizure-induced c-fos expression in rat medulla oblongata is not dependent on associated elevation of blood pressure
TL;DR: Immunohistochemical study of sections of medulla oblongata revealed that seizures are followed by induction of c-fos IR in nucleus tractus solitarius (NTS), dorsal motor nucleus of the vagus (DMN 10), and ventrolateral medulla (VLM), while there is negligible c- fos IR after saline sham injections.
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Retinoic acid induces translocation of protein kinase C (PKC) and activation of nuclear PKC (nPKC) in rat splenocytes
Nancy E. Zorn,Marie D. Sauro +1 more
TL;DR: The ability of RA to activate cell membrane PKC resulting in an increase in particulate PKC activity correlates well with the activation of nPKC since the particulate fraction would include nuclear enzyme systems.
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