Manar A. Nader
Mansoura University
56 Papers
68 Citations
Manar A. Nader is an academic researcher from Mansoura University. The author has contributed to research in topics: Oxidative stress & Chemistry. The author has an hindex of 13, co-authored 42 publications. Previous affiliations of Manar A. Nader include Taibah University.
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Papers
Sitagliptin enhances the neuroprotective effect of pregabalin against pentylenetetrazole-induced acute epileptogenesis in mice: Implication of oxidative, inflammatory, apoptotic and autophagy pathways
TL;DR: This study is indicative for the antiepileptogenic potential of sitagliptin with or without pregabalin in the PTZ model of epilepsy which is likely to be through its effect on antioxidant, anti‐apoptotic and autophagic pathways.
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Effect of betulinic acid on neutrophil recruitment and inflammatory mediator expression in lipopolysaccharide-induced lung inflammation in rats.
Manar A. Nader,Hany N. Baraka +1 more
TL;DR: The present results suggest that BA is endowed with antiinflammatory and antioxidant properties that protect the lung against the deleterious actions of LPS.
50
Sitagliptin exerts anti-inflammatory and anti-allergic effects in ovalbumin-induced murine model of allergic airway disease
TL;DR: Sitagliptin attenuates the allergic airway inflammation suggesting that sitagli leptin may have applications in the treatment of bronchial asthma.
39
Tiron protects against nicotine-induced lung and liver injury through antioxidant and anti-inflammatory actions in rats in vivo.
TL;DR: Tiron (synthetic analogue of vitamin E) good candidate for future use to minimize nicotine's hazards among smokers as proved by a significant decrease in malondialdehyde content, reactive oxygen species (ROS) and total nitrite/nitrate (NOx) levels, and increase in reduced glutathione (GSH) level.
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Attenuation of oxidative stress-induced vascular endothelial dysfunction by thymoquinone.
Dina S. El-Agamy,Manar A. Nader +1 more
TL;DR: The results suggest that TQ can protect against pyrogallol-induced endothelial dysfunction which probably results from its potent antioxidant capacity that leads to an increase in NO production as well as its ability to enhance the generation and bioavailability of NO.
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