Maki Ishii
Osaka Prefecture University
5 Papers
37 Citations
Maki Ishii is an academic researcher from Osaka Prefecture University. The author has contributed to research in topics: Ephrin & Erythropoietin-producing hepatocellular (Eph) receptor. The author has an hindex of 5, co-authored 5 publications.
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Papers
EphB signaling inhibits gap junctional intercellular communication and synchronized contraction in cultured cardiomyocytes
Maki Ishii,Itsuki Mueller,Takayuki Nakajima,Elena B. Pasquale,Elena B. Pasquale,Kazushige Ogawa +5 more
TL;DR: The present findings suggest that ephrin-B-EphB signaling can modulate the electrical coupling of cardiomyocytes through effects on gap junctions.
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Effective and Steady Differentiation of a Clonal Derivative of P19CL6 Embryonal Carcinoma Cell Line into Beating Cardiomyocytes
TL;DR: It is demonstrated that the correlograms could clearly display the enhancement of beating rates by cardiotonic reagents, indicating that a combination of P19CL6-A1 and Visorhythm is a useful tool that can provide invaluable assistance in inotropic drug discovery, drug screening, and toxicity testing.
Complementary expression and repulsive signaling suggest that EphB receptors and ephrin-B ligands control cell positioning in the gastric epithelium
Kazushige Ogawa,Natsuki Takemoto,Maki Ishii,Elena B. Pasquale,Elena B. Pasquale,Takayuki Nakajima +5 more
TL;DR: It is proposed that the EphB-positive progeny of gastric stem cells migrates from the isthmus toward the bottom of the gastric glands due to repulsive signals arising from contact with ephrin-Bs, which are preferentially expressed in the more superficial regions of the isThmus and gastric pits.
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Ischemic preconditioning attenuates of ischemia-induced degradation of spectrin and tau: implications for ischemic tolerance
TL;DR: The results suggest that the attenuation of ischemia-induced degradation of α-spectrin and tau by ischemic preconditioning may be associated with the neuroprotective mechanism of the isChemic tolerance.
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Complementary expression of EphB receptors and ephrin-B ligand in the pyloric and duodenal epithelium of adult mice
TL;DR: It is proposed that EphB/ephrin-B signalling may regulate cell positioning within the pyloric and duodenal epithelium.
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