Maisha Minor
University of Colorado Denver
4 Papers
19 Citations
Maisha Minor is an academic researcher from University of Colorado Denver. The author has contributed to research in topics: Regulation of gene expression & Tumor necrosis factor alpha. The author has an hindex of 4, co-authored 4 publications.
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Papers
Cigarette Smoke Increases Susceptibility to Tuberculosis—Evidence From In Vivo and In Vitro Models
Shaobin Shang,Diane J. Ordway,Marcela Henao-Tamayo,Xiyuan Bai,Rebecca E. Oberley-Deegan,Crystal A. Shanley,Ian M. Orme,Stephanie Case,Maisha Minor,David F. Ackart,Laurel Hascall-Dove,Alida R. Ovrutsky,Pitchaimani Kandasamy,Dennis R. Voelker,Cherie Lambert,Brian M. Freed,Michael D. Iseman,Randall J. Basaraba,Edward D. Chan,Edward D. Chan +19 more
TL;DR: CS suppresses the protective immune response to M. tuberculosis in mice, human THP-1 cells, and primary human alveolar macrophages, and humans exposed to CS extract, nicotine, or acrolein.
SPLUNC1 regulation in airway epithelial cells: role of toll-like receptor 2 signaling
Hong Wei Chu,Fabienne Gally,Jyoti Thaikoottathil,Yvonne M. W. Janssen-Heininger,Qun Wu,Gongyi Zhang,Nichole Reisdorph,Stephanie Case,Maisha Minor,Sean Smith,Di Jiang,Nicole Michels,Glenn C. Simon,Richard J. Martin +13 more
TL;DR: Data for the first time suggest that airway epithelial TLR2 signaling is pivotal in mycoplasma-induced SPLUNC1 production, thus improving the understanding of the aberrant SPLunC1 expression in airways of patients suffering from chronic lung diseases with bacterial infections.
Airway Epithelial NF-κB Activation Promotes Mycoplasma pneumoniae Clearance in Mice
Di Jiang,Mark L. Nelson,Fabienne Gally,Sean Smith,Qun Wu,Maisha Minor,Stephanie Case,Jyoti Thaikoottathil,Hong Wei Chu +8 more
TL;DR: In vivo airway epithelial NF-κB activation promotes lung bacterial clearance, which is accompanied by increased epithelial SPLUNC1 expression, and is demonstrated by using the non-antimicrobial 9-TB.
Roflumilast increases Clara cell secretory protein in cigarette smoke-exposed mice.
TL;DR: It is demonstrated that one of the therapeutic mechanisms of roflumilast is to reverse CS-induced downward trend in CCSP levels of BAL fluid, which may be mediated by down-regulating ERK1/2 activity.
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