Maarten Naesens
Katholieke Universiteit Leuven
294 Papers
768 Citations
Maarten Naesens is an academic researcher from Katholieke Universiteit Leuven. The author has contributed to research in topics: Transplantation & Medicine. The author has an hindex of 48, co-authored 221 publications. Previous affiliations of Maarten Naesens include The Catholic University of America & California Pacific Medical Center.
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Papers
Subclinical peritubular capillaritis at 3 months is associated with chronic rejection at 1 year.
TL;DR: Through early detection of subclinical peritubular capillaritis, renal allograft recipients who are at risk for development of MLPTC might be identified.
A Practical Guide to the Clinical Implementation of Biomarkers for Subclinical Rejection Following Kidney Transplantation.
TL;DR: An overview of the process by which the incremental value of each biomarker is evaluated to determine its potential role in clinical practice is provided, providing a framework that can be used by clinicians as a practical guide in general and more specifically in the context of subclinical rejection following KT.
Urinary Protein Biomarker Panel for the Diagnosis of Antibody-Mediated Rejection in Kidney Transplant Recipients.
Inge Mertens,Hanny Willems,Elisabet Van Loon,Karin Schildermans,Kurt Boonen,Geert Baggerman,Dirk Valkenborg,Wilfried Gwinner,Dany Anglicheau,Marie Essig,Pierre Marquet,Maarten Naesens +11 more
TL;DR: This study identified and validated a urinary protein biomarker set that can be used to exclude Antibody-mediated rejection after kidney transplantation and found it to be better than the accuracy of gross proteinuria.
Microscopic nephrocalcinosis in chronic kidney disease patients
Pieter Evenepoel,Kristien Daenen,Bert Bammens,Kathleen Claes,Björn Meijers,Maarten Naesens,Ben Sprangers,Dirk Kuypers,Eveline Lerut +8 more
TL;DR: The data demonstrate that prevalence rates of nephrocalcinosis increase with increasing CKD stage to reach more than 50% in end-stage renal disease patients and suggest that acid-base and mineral metabolism disturbances are implicated in its pathogenesis.
Time-dependent variations in BK polyomavirus genome from kidney transplant recipients with persistent viremia
Olga Mineeva-Sangwo,Elisabet Van Loon,Graciela Andrei,Dirk Kuypers,Maarten Naesens,Robert Snoeck +5 more
TL;DR: It is shown that mutations occur throughout the entire BKPyV genome, including in VP1, and that mutations can accumulate over time in the major capsid protein VP1 and can lead to neutralization escape in kidney transplant recipients.