M. Michael Glovsky
California Institute of Technology
41 Papers
710 Citations
M. Michael Glovsky is an academic researcher from California Institute of Technology. The author has contributed to research in topics: Pollen & Complement system. The author has an hindex of 20, co-authored 41 publications. Previous affiliations of M. Michael Glovsky include Huntington Hospital.
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Papers
C5a-induced expression of P-selectin in endothelial cells.
Kimberly E. Foreman,Ara A. Vaporciyan,Brian Bonish,Michael L. Jones,Kent J. Johnson,Kent J. Johnson,M. Michael Glovsky,Susan M. Eddy,Peter A. Ward +8 more
TL;DR: Data suggest that C5a may be an important inflammatory mediator for the early adhesive interactions between neutrophils and endothelial cells in the acute inflammatory response.
C5a-induced Expression ofP-selectin inEndothelial Cells
Kimberly E. Foreman,BrianK . Bonish,Michael L. Jones,KentJ . Johnson,M. Michael Glovsky,SusanM . Eddy +5 more
- 01 Jan 1994
TL;DR: A cell-based ELISA technique has confirmed upregulation of P-selectin in endothelial cells exposed to C5a as mentioned in this paper, withmolecules bound being - 10%ofthose binding toneutrophils.
421
Release of allergens as respirable aerosols: A link between grass pollen and asthma
TL;DR: First direct observations of the release of grass pollen allergens as respirable aerosols can emanate directly from the flower after a moisture-drying cycle are provided, which could explain asthmatic responses associated with grass pollination, particularly after moist weather conditions.
287
Allergens in paved road dust and airborne particles
TL;DR: In this article, the authors used immunological assays that measure the proteins extracted from environmental samples that bind to IgE antibodies present in the blood serum of allergenic patients to measure the allergen concentrations present in paved road dust and in airborne particle samples.
140
Links between pollen, atopy and the asthma epidemic.
TL;DR: This review outlines the mechanism of pollen fragmentation and possible pathophysiology of pollen fragment-induced asthma and examines synergistic effects and enhanced immune response from interaction in the atmosphere, or from co-deposition in the airways, of pollen allergens, endogenous pro-inflammatory agents, and the particulate and gaseous fraction of combustion products.