M. Madsen
Aarhus University Hospital
22 Papers
126 Citations
M. Madsen is an academic researcher from Aarhus University Hospital. The author has contributed to research in topics: Transplantation & Organ donation. The author has an hindex of 7, co-authored 22 publications.
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Papers
C2 (2‐h) levels are not superior to trough levels as estimates of the area under the curve in tacrolimus‐treated renal‐transplant patients
Kaj Anker Jørgensen,Johan V. Povlsen,Søren Madsen,M. Madsen,Hans Erik Hansen,Asger Pedersen,Else-Marie Heinsvig,Jørgen H. Poulsen +7 more
TL;DR: Two-hour levels are not superior to trough levels in tacrolimus-treated renal transplant patients, and despite good correlation between trough level and AUC, some patients may still receive nephrotoxic doses despite trough level in the desired range.
Decreased expression of HLA-DR antigens on peripheral blood B lymphocytes during glucocorticoid treatment.
TL;DR: It is concluded that glucocorticoid administration in vivo decreases the expression of HLA-DR antigens on PBBL, possibly in an indirect way, the mechanism of which is still unknown.
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Chronic cyclosporine nephrotoxicity: a pig model.
Donata Cibulskyte,H. Kaalund,Michael Pedersen,Arne Hørlyck,Niels Marcussen,Hans Erik Hansen,M. Madsen,Jens Tølbøll Mortensen +7 more
- 01 Oct 2005
TL;DR: It is concluded that the pig model displays a hyperfiltration that warrants further investigation, and shows that the whole blood trough CsA levels were lower in pigs than in humans treated with similarCsA doses.
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Organ donation, allocation, and transplantation in the Nordic countries: Scandiatransplant 1999.
TL;DR: Organ donation, allocation, and transplantation in the Nordic countries: Scandiatransplant 1999.
11
Does Chronic Low-Dose Treatment With Cyclosporine Influence the Brain? A Histopatological Study in Pigs
Frederikke Rosendal,Carsten R. Bjarkam,Mette Bach Larsen,Hans Erik Hansen,M. Madsen,Jens Christian Sørensen,Jens Tølbøll Mortensen +6 more
- 01 Oct 2005
TL;DR: Pathological changes were noticed in one out of five CsA-treated animals, corresponding to the percentage of patients treated with Cs a who develop CsB encephalopathy, to elucidate histopathological changes in the brain.
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