Long Li
Ningbo University
11 Papers
24 Citations
Long Li is an academic researcher from Ningbo University. The author has contributed to research in topics: Chemistry & Pyroptosis. The author has an hindex of 4, co-authored 6 publications.
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Papers
Pyroptosis of syncytia formed by fusion of SARS-CoV-2 spike and ACE2-expressing cells.
Huabin Ma,Huabin Ma,Zhoujie Zhu,Huaipeng Lin,Shanshan Wang,Peipei Zhang,Yanguo Li,Long Li,Jinling Wang,Yufen Zhao,Jiahuai Han +10 more
TL;DR: In this article, the authors established an in vitro cell-cell fusion system and used it to mimic the fusion of SARS-CoV-2 infected cells with ACE2-expressing cells to form syncytia.
Oleoylethanolamide Protects Against Acute Liver Injury by Regulating Nrf-2/HO-1 and NLRP3 Pathways in Mice.
TL;DR: In this article, the effects and mechanisms of OEA in lipopolysaccharide (LPS)/d-galactosamine (D-Gal)-induced acute liver injury in mice were investigated.
Micronized Palmitoylethanolamide Ameliorates Methionine- and Choline-Deficient Diet-Induced Nonalcoholic Steatohepatitis via Inhibiting Inflammation and Restoring Autophagy.
TL;DR: In this paper, the effect of PEA on nonalcoholic steatohepatitis (NASH) was explored and the underlying mechanism was revealed, which suggested that PEA protects against NASH through the inhibition of inflammation and restoration of autophagy.
Natural Potent NAAA Inhibitor Atractylodin Counteracts LPS-Induced Microglial Activation.
Longhe Yang,Chunyan Ji,Yitian Li,Fan Hu,Fang Zhang,Haiping Zhang,Long Li,Jie Ren,Zhaokai Wang,Yan Qiu +9 more
TL;DR: Results show that atractylodin elevates cellular PEA levels and inhibits microglial activation by inhibiting NAAA activity, which in turn could contribute to NAAA functional research.
Protein phosphorylation and kinases: Potential therapeutic targets in necroptosis.
Chengkun Wu,Jiayi Shi,Taotao Gao,Huabin Ma,Long Li,Yufen Zhao +5 more
TL;DR: This review explores the role of protein phosphorylation and kinases in necroptosis, a cell death pathway implicated in various diseases, and discusses the potential of kinase inhibitors as therapeutic targets for modulating necroptotic processes and treating diseases such as cancer.
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