Liping Wang
University of California, San Francisco
12 Papers
Liping Wang is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Chemistry & Osteoblast. The author has an hindex of 8, co-authored 12 publications. Previous affiliations of Liping Wang include Chulalongkorn University & San Francisco VA Medical Center.
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Papers
Investigating the cellular origin of rotator cuff muscle fatty infiltration and fibrosis after injury.
Xuhui Liu,Anne Y. Ning,Nai Chen Chang,Hubert T. Kim,Robert A. Nissenson,Liping Wang,Brian T. Feeley +6 more
TL;DR: Data demonstrated that the Tie2(+) muscle mesenchymal progenitors are the major source of fibroblasts while PDGFRα(+) FAPs are theMajor source of adipocytes in rotator cuff muscle fatty infiltration.
Estrogen signaling in arcuate Kiss1 neurons suppresses a sex-dependent female circuit promoting dense strong bones.
Candice B. Herber,William C. Krause,Liping Wang,James R. Bayrer,Alfred Li,Matthew T. Schmitz,Aaron J. Fields,Breanna Ford,Zhi Zhang,Michelle S. Reid,Daniel K. Nomura,Robert A. Nissenson,Stephanie M. Correa,Stephanie M. Correa,Holly A. Ingraham +14 more
TL;DR: The authors show that loss of estrogen signalling after ablating estrogen receptor alpha (ERa) in specific hypothalamic neuronal populations leads to a marked sex-dependent increase in bone mass in female mice, revealing a previously unknown target for treatment of age-related bone disease.
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A novel mouse model of trauma induced heterotopic ossification
Xuhui Liu,Heejae Kang,Mohammad Shahnazari,Mohammad Shahnazari,Hubert T. Kim,Hubert T. Kim,Liping Wang,Liping Wang,Olla Larm,Lars Adolfsson,Robert A. Nissenson,Robert A. Nissenson,Bernard P. Halloran,Bernard P. Halloran +13 more
TL;DR: It is suggested that BMP‐2 signaling can sensitize muscle to trauma‐induced HO, and this model provides the basis for a new model to study the pathogenesis of trauma‐ induced HO.
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Increased Bone Mass in Mice Lacking the Adipokine Apelin
Lalita Wattanachanya,W Lu,Ramendra K. Kundu,Liping Wang,Liping Wang,Marcia J. Abbott,Dylan O'Carroll,Thomas Quertermous,Robert A. Nissenson +8 more
TL;DR: The increased bone mass in mice lacking apelin suggested complex direct and paracrine/endocrine effects of apelin on bone, possibly via modulating insulin sensitivity, indicating that apelin functions as a physiologically significant antianabolic factor in bone in vivo.
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Osteoblast-derived FGF9 regulates skeletal homeostasis.
Liping Wang,Theresa M. Roth,Marcia J. Abbott,Linh Ho,Lalita Wattanachanya,Robert A. Nissenson,Robert A. Nissenson +6 more
TL;DR: The results suggest that mature OBs are an important source of FGF9, positively regulating skeletal homeostasis in male mice, and may serve a paracrine role to maintain the osteogenic progenitor cell population through activation of Akt signaling.
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