A scoping review of the health and toxicological activity of bisphenol A (BPA) structural analogues and functional alternatives

Peroxymonosulfate activation by porous BiFeO3 for the degradation of bisphenol AF: Non-radical and radical mechanism

Bisphenol AF compromises blood-testis barrier integrity and sperm quality in mice.

Replacement Bisphenols Adversely Affect Mouse Gametogenesis with Consequences for Subsequent Generations

Bisphenol A (BPA), a compound used in the manufacturing of plastics and epoxy resins, is an endocrine disruptor with significant adverse impact on the human's health. Here, we review the animal models and clinical studies as well as the molecular and cellular mechanisms that show that BPA alters the normal function of the reproductive system, metabolism, brain function and behavior and contributes to the development of certain neurodevelopmental disorders including autism spectrum and attention-deficit and hyperactivity disorders. BPA also causes aberrant cognitive function, behavioral disturbances, and neurodegenerative diseases, including Parkinson's disease, amyotrophic lateral sclerosis (ALS), and multiple sclerosis. It has recently been proposed that exposure to BPA may be associated with the development of certain neurodegenerative diseases and neurodevelopmental disorders; however, it is a line of research that is just emerging. This work aims to review the available information about the association between exposure to BPA and cognitive function, behavioral disturbances, neurodegenerative diseases (Parkinsoni?½s Disease, Amyotrophic lateral sclerosis, Multiple Sclerosis), and neurodevelopmental disorders (Autism Spectrum and Attention-Deficit/Hyperactivity Disorders). Likewise, the molecular and cellular mechanisms that may be involved with these pathological conditions will be analyzed.

Impact of BPA on behavior, neurodevelopment and neurodegeneration

Effects of maternal exposure to bisphenol AF on emotional behaviors in adolescent mice offspring

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Effects of maternal exposure to bisphenol AF on emotional behaviors in adolescent mice offspring