Linda E. Pelinka
University of Vienna
34 Papers
322 Citations
Linda E. Pelinka is an academic researcher from University of Vienna. The author has contributed to research in topics: Intensive care & Traumatic brain injury. The author has an hindex of 20, co-authored 34 publications.
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Papers
GFAP versus S100B in serum after traumatic brain injury: relationship to brain damage and outcome.
TL;DR: Both measurement of GFAP and S100B is a useful non-invasive means of identifying brain damage with some differences based on the pattern of TBI and accompanying multiple trauma and/or shock.
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Glial fibrillary acidic protein in serum after traumatic brain injury and multiple trauma.
Linda E. Pelinka,Alfred Kroepfl,Robert Schmidhammer,Manfred Krenn,Walter Buchinger,Heinz Redl,Andreas Raabe +6 more
TL;DR: The findings showed that GFAP is released after TBI, thatGFAP is related to brain injury severity and outcome after T BI, and that GF AP is not released after multiple trauma without brain injury.
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Serum S 100 B: a marker of brain damage in traumatic brain injury with and without multiple trauma.
TL;DR: Though S 100 B may be a reliable marker of brain damage in TBI without multiple trauma 24 h after trauma and thereafter, it appears to be less reliable in TBA with multiple trauma.
129
Elevated serum levels of epithelial cell apoptosis-specific cytokeratin 18 neoepitope m30 in critically ill patients.
Georg A. Roth,Claus G. Krenn,Markus Brunner,Bernhard Moser,Martin Ploder,Andreas Spittler,Linda E. Pelinka,Thomas Sautner,Ernst Wolner,George Boltz-Nitulescu,Hendrik Jan Ankersmit +10 more
TL;DR: The increased serum level of the CK18 neoepitope in septic patients indicates a heightened apoptotic turnover in epithelial cells as compared with trauma patients and healthy controls and interestingly, nonsurviving trauma patients exhibited a significant increase in the M30 neoantigen.
84
Circulating S100B is increased after bilateral femur fracture without brain injury in the rat
Linda E. Pelinka,László Szalay,Mohammad Jafarmadar,Robert Schmidhammer,Heinz Redl,Soheyl Bahrami +5 more
TL;DR: S100B is increased in bilateral femur fracture without haemorrhagic shock in rats, and this finding suggests that bone marrow is a potential extracerebral source of S100B.
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