Lin Li
5 Papers
8 Citations
Lin Li is an academic researcher. The author has contributed to research in topics: Phosphorylation & Medicine. The author has an hindex of 2, co-authored 2 publications.
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Papers
Creatine kinase B suppresses ferroptosis by phosphorylating GPX4 through a moonlighting function
Ke Wu,Meisi Yan,Tong-Xin Liu,Zheng-Hua Wang,Yu-Qiu Duan,Yan Xia,Guimei Ji,Yuling Shen,Lei Wang,Lin Li,Peixiang Zheng,Bofei Dong,Qingang Wu,Liwei Xiao,Xueying Yang,Hao-Miao Shen,Jingjing Zhang,Jinfeng Yi,Yu-Yi Deng,Xu Qian,Leina Ma,Jing Fang,Qingxin Zhou,Zhimin Lu,Daqian Xu +24 more
TL;DR: Findings reveal a critical mechanism by which tumour cells counteract ferroptosis by non-metabolic function of C KB-enhanced GPX4 stability and underscore the potential to target the protein kinase activity of CKB for cancer treatment.
75
Fructose-1,6-bisphosphatase 1 functions as a protein phosphatase to dephosphorylate histone H3 and suppresses PPARα-regulated gene transcription and tumour growth
Zheng-Hua Wang,Min Li,Hongfei Jiang,Shudi Luo,Fei Shao,Yan Xia,Meng-qing Yang,Xiangle Ren,Tong-Xin Liu,Meisi Yan,Xu Qian,Haiyan He,Dong Guo,Yu-Qiu Duan,Ke Wu,Lei Wang,Guimei Ji,Yu-liang Shen,Lin Li,Peixiang Zheng,Bofei Dong,Jing Fang,Min Zheng,Tingbo Liang,Haitao Li,Rilei Yu,Daqian Xu,Zhimin Lu +27 more
TL;DR: It is demonstrated that glucose deprivation in normal hepatocytes induces PERK-mediated fructose-1,6-bisphosphatase 1 (FBP1) S170 phosphorylation, which converts the FBP1 tetramer to monomers and exposes its nuclear localization signal for nuclear translocation.
32
Computationally Sound Mechanized Proofs for Electronic Payment Protocol in a Probabilistic Polynomial Calculus with CryptoVerif
TL;DR: The result shows that SOCPT electronic payment protocol has money accountability and goods accountability, which is consistent with its claim, and the first automatic analysis of SOCPT Electronic payment protocol in computational model is conducted.
2
Nucleus-exported CLOCK acetylates PRPS to promote de novo nucleotide synthesis and liver tumour growth
Tong Liu,Zheng-Hua Wang,Leiguang Ye,Yu-Qiu Duan,Hongfei Jiang,Haiyan He,Liwei Xiao,Qingang Wu,Yan Xia,Meng-qing Yang,Ke Wu,Meisi Yan,Guimei Ji,Yuling Shen,Lei Wang,Lin Li,Peixiang Zheng,Bofei Dong,Fei Shao,Xu Qian,Rilei Yu,Zhiren Zhang,Zhimin Lu,Daqian Xu +23 more
TL;DR: A critical mechanism by which oncogenic signalling inhibits canonical CLOCK transcriptional activity and simultaneously confers CLOCK with instrumental moonlighting functions to promote nucleotide synthesis and tumour growth is delineated.