Li Li
Southwest University
53 Papers
64 Citations
Li Li is an academic researcher from Southwest University. The author has contributed to research in topics: Zebrafish & Apoptosis. The author has an hindex of 15, co-authored 44 publications. Previous affiliations of Li Li include Hong Kong University of Science and Technology & Georgetown University.
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Papers
Live imaging reveals differing roles of macrophages and neutrophils during Zebrafish tail fin regeneration
TL;DR: It is shown that macrophages and neutrophils play distinct functions in tissue regeneration, efficiently resolving inflammation and facilitating tissue remodeling and regrowth.
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cMyb regulates hematopoietic stem/progenitor cell mobilization during zebrafish hematopoiesis.
TL;DR: This study reveals that cMyb plays a hitherto unidentified role in dictating physiologic HSPC migration by modulating Sdf1a signaling.
93
Il-1β and Reactive Oxygen Species Differentially Regulate Neutrophil Directional Migration and Basal Random Motility in a Zebrafish Injury–Induced Inflammation Model
Bo Yan,Peidong Han,Lifeng Pan,Wei Lu,Jing-Wei Xiong,Mingjie Zhang,Wenqing Zhang,Li Li,Zilong Wen +8 more
TL;DR: It is revealed that the Il-1β–Myd88 axis and NADPH oxidase–mediated ROS signaling are two independent pathways that differentially regulate neutrophil migration during sterile inflammation.
86
Sensitization of tamoxifen-resistant breast cancer cells by Z-ligustilide through inhibiting autophagy and accumulating DNA damages
TL;DR: Findings provide important insights into the formation of tamoxifen resistance in breast cancer cells, but also suggest Z-ligustilide may function as a novel autophagy inhibitor to overcome chemoresistance.
Distinct regulatory networks control the development of macrophages of different origins in zebrafish.
TL;DR: It is shown that RBI- and VDA-born macrophages are orchestrated by distinctive regulatory networks formed by the E-twenty-six (Ets) transcription factors Pu.1 and Spi-b, the zebrafish ortholog of mouse spleen focus forming virus proviral integration oncogene B (SPI-B), and the helix-turn-helix DNA-binding domain containing protein Irf8.1.
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