Lehel Peterfi
University of Pécs
12 Papers
13 Citations
Lehel Peterfi is an academic researcher from University of Pécs. The author has contributed to research in topics: Medicine & Tissue microarray. The author has an hindex of 4, co-authored 9 publications.
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Papers
Expression of RARRES1 and AGBL2 and progression of conventional renal cell carcinoma.
TL;DR: It is found that RAR RES1 cytoplasmic/negative and AGBL2-positive/negative staining is a significant risk factor for tumour progression indicating 11–15 times higher risk of cancer relapse, whereas the membranous RARRES1 expression, especially its co-expression with AGBl2, associated with excellent disease outcome.
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Dual role of KRT17: development of papillary renal cell tumor and progression of conventional renal cell carcinoma.
TL;DR: The continuous expression of KRT17 from emerging fetal kidney tubules and microscopic pre-neoplastic lesions towards papillary renal cell tumors and its neo-expression in aggressive growing conventional renal cell carcinomas reflects the multiple function of K RT17 in kidney cancers with distinct natural history.
Expression of TXNIP is associated with angiogenesis and postoperative relapse of conventional renal cell carcinoma
Mate Meszaros,Maria V. Yusenko,Lilla Domonkos,Lehel Peterfi,Gyula Kovacs,Gyula Kovacs,Daniel Banyai +6 more
TL;DR: In this paper, the role of TXNIP and downstream genes HIF1α and IL1β in the biology of conventional RCC (cRCC) was investigated and a significant correlation was found between cytoplasmicTXNIP expression, inefficient vascularization by unorganized and tortuous vessels causing tumour cell necrosis and postoperative tumour relapse of cRCC.
Expression of inflammatory lipopolysaccharide binding protein (LBP) predicts the progression of conventional renal cell carcinoma - a short report.
TL;DR: LBP expression in conventional RCC defines a group of patients at a high risk of postoperative progression and may help to direct optimized active surveillance and timely adjuvant therapy.
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IL6 Shapes an Inflammatory Microenvironment and Triggers the Development of Unique Types of Cancer in End-stage Kidney.
TL;DR: The data indicate that expression of IL6, acute phase protein SAA1 and LBP maintain a long-lasting inflammatory microenvironment that leads to remodeling of end-stage kidneys and the development of unique types of renal cell tumors.
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