Lanhao Liu
Qingdao Agricultural University
14 Papers
5 Citations
Lanhao Liu is an academic researcher from Qingdao Agricultural University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 1, co-authored 2 publications.
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Papers
Identification of type I IFNs and their receptors in a cyprinid fish, the topmouth culter Culter alburnus
TL;DR: Four type I IFNs were identified in a cyprinid, the topmouth culter, Culter alburnus, a species introduced recently into China's aquaculture and the possible function of these IFNs and their signalling pathway are of interest for further research.
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Molecular identification and function analysis of bactericidal permeability-increasing protein/LPS-binding protein 1 (BPI/LBP1) from turbot (Scophthalmus maximus).
TL;DR: The results indicated that SmBPI/LBP1 probably played important roles in immune response against bacteria infection, and was ubiquitously expressed in all tested tissues, with the highest expression level in spleen tissue.
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MicroRNA miR-722 Inhibits Cyprinid Herpesvirus 3 Replication via Targeting the Viral Immune Evasion Protein ORF89, Which Negatively Regulates IFN by Degrading IRF3.
TL;DR: Zhang et al. as discussed by the authors found that microRNA (miR)-722 was upregulated during CyHV-3 infection, indicating that miR-722 might play an important role in the pathogenicity of cyprinid herpesvirus 3.
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Identification of type II interferons and receptors in an osteoglossiform fish, the arapaima Arapaima gigas.
An Ning Pang,Shan Nan Chen,Zhen Gan,Li Li,Nan Li,Shuai Wang,Zhenglong Sun,Lanhao Liu,Yanrong Sun,Xiao-Jun Song,Yang Liu,Su Wang,Pin Nie +12 more
TL;DR: In this article , arapaima Arapaimaima gigas, three type II interferon (IFN) genes were identified in an osteoglossiform fish.
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MicroRNA miR-722 Inhibits Cyprinid Herpesvirus 3 Replication via Targeting the Viral Immune Evasion Protein ORF89, Which Negatively Regulates IFN by Degrading IRF3
TL;DR: These findings confirm a novel virus-host combat, in which CyHV-3 evades host antiviral immunity by its ORF89 protein, whereas host miR-722, upregulated on CyHv-3 infection, targets ORF 89 to impede CyH V-3 replication.
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