Kui-Sheng Chen
Zhengzhou University
39 Papers
118 Citations
Kui-Sheng Chen is an academic researcher from Zhengzhou University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 8, co-authored 23 publications.
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Papers
New insights into the interplay between long non‐coding RNAs and RNA‐binding proteins in cancer
TL;DR: How the lncRNA‐RBP interaction network regulates cancer cell phenotypes, such as proliferation, apoptosis, metastasis, drug resistance, immunity, tumor environment, and metabolism, is discussed and the therapeutic strategies that target this network are summarized.
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Correlation of matrix metalloproteinase suppressor genes RECK, VEGF, and CD105 with angiogenesis and biological behavior in esophageal squamous cell carcinoma
Shenglei Li,Dongling Gao,Zhi-Hua Zhao,Zong-Wen Liu,Qiu-Min Zhao,Jin-Xia Yu,Kui-Sheng Chen,Yunhan Zhang +7 more
TL;DR: Correlation of matrix metalloproteinase suppressor genes RECK, VEGF, and CD105 with angiogenesis and biological behavior in esophageal squamous cell carcinoma was found in this paper.
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Exosomal miR-301a-3p from esophageal squamous cell carcinoma cells promotes angiogenesis by inducing M2 polarization of macrophages via the PTEN/PI3K/AKT signaling pathway
Yuwei Shou,Xiaoqian Wang,Chao Chen,Yinghao Liang,Chenbo Yang,Qiankun Xiao,Hui Li,Shuaiyu Wang,Jiaojie Shu,Xiangyu Tian,Kui-Sheng Chen +10 more
TL;DR: In this paper , exosomal miR-301a-3p secreted by esophageal squamous cell carcinoma (ESCC) cells triggers the pro-angiogenic switch of TAMs.
•Journal Article
ZEB1 promotes the progression and metastasis of cervical squamous cell carcinoma via the promotion of epithelial-mesenchymal transition.
TL;DR: The upregulation of ZEB1 is associated with the abnormal expression of E-cadherin, β-catenin and N-cadaverin, which might promote the progression and metastasis of cervical squamous cell carcinoma.
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Exosomes-derived miR-154-5p attenuates esophageal squamous cell carcinoma progression and angiogenesis by targeting kinesin family member 14
TL;DR: It is identified that exosomes-derived miR-154-5p attenuates ESCC progression and angiogenesis by targeting KIF14 in vitro, which might provide a novel approach for the diagnosis and treatment of ESCC.
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