Krzysztof Moroz
Tulane University
62 Papers
274 Citations
Krzysztof Moroz is an academic researcher from Tulane University. The author has contributed to research in topics: Cancer & Metastasis. The author has an hindex of 21, co-authored 57 publications.
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Papers
Somatic mutations in the DNA repairome in prostate cancers in African Americans and Caucasians.
Santosh Yadav,Muralidharan Anbalagan,Melody Baddoo,Vinodh K. Chellamuthu,Sudurika Mukhopadhyay,Carol Woods,Wei Jiang,Krzysztof Moroz,Erik K. Flemington,Nick M. Makridakis +9 more
TL;DR: Tumors in Caucasians did not show a correlation with age, but a progressive increase in the mutation rate was observed at higher Gleason scores, and a marginal increase in mutation rate in tumors in African Americans with increasing age was identified.
Examining the Bethesda criteria risk stratification of thyroid nodules
TL;DR: The current Bethesda risk stratification system underestimated malignancy rates in benign, indeterminate and non-diagnostic cytopathologic categories in the authors' experience and there was no positive linear correlation between nodule size and malignancies rate in these cytopATHologic categories.
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In vivo anticancer synergy mechanism of doxorubicin and verapamil combination treatment is impaired in BALB/c mice with metastatic breast cancer
Michelle McCarthy,Gregory Auda,Suchi Agrawal,Amy Taylor,Zack Backstrom,Debasis Mondal,Krzysztof Moroz,Srikanta Dash +7 more
TL;DR: It is proposed that understanding the mechanisms as to why the combination of doxorubicin and verapamil treatment was impaired in the mouse model should allow novel approaches to improve chemotherapy response of metastatic breast cancer.
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Immunolocalization of DCAMKL-1, a putative intestinal stem cell marker, in normal colonic tissue.
TL;DR: The findings suggest that DCAMKL-1 marks a subset of colorectal stem cells, as well as a subsets of enteroendocrine cells in normal colonic mucosa.
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Chaperone-Mediated Autophagy in the Liver: Good or Bad?
TL;DR: It is proposed that a better understanding of how excessive cellular stress leads to cancer through autophagy modulation may allow therapeutic development and early detection of HCC.
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