Klaus Berger
University of Münster
491 Papers
2.6K Citations
Klaus Berger is an academic researcher from University of Münster. The author has contributed to research in topics: Medicine & Population. The author has an hindex of 84, co-authored 428 publications. Previous affiliations of Klaus Berger include Erasmus University Rotterdam & University of Duisburg-Essen.
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Papers
Quality of care of acute ischemic stroke in Germany 2018
Björn Misselwitz,A Grau,Klaus Berger,I Bruder,C Burmeister,Peter Hermanek,R Hohnhold,H C Koennecke,C. Matthis,Peter U. Heuschmann +9 more
TL;DR: The results regarding the defined 16 quality indicators showed a high level of acute inpatient treatment of stroke in Germany and only a few quality indicators, such as early transfer for thrombectomy indicated a great necessity for process optimization.
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Stratified prevention: opportunities and limitations. Report on the 1st interdisciplinary cardiovascular workshop in Augsburg.
Gregor Kirchhof,Josef Franz Lindner,Stephan Achenbach,Klaus Berger,Stefan Blankenberg,Heiner Fangerau,Henner Gimpel,Ulrich M. Gassner,Jens Kersten,Dorothea Magnus,Herbert Rebscher,Heribert Schunkert,Stephan Rixen,Paulus Kirchhof +13 more
TL;DR: Stratified prevention requires the evidence-based development of a new taxonomy of cardiovascular diseases that reflects disease mechanisms and needs broad support from society and a better use of biosamples and data sets within an updated research governance framework.
Investigating the roles of age, sex, depression, and anxiety for valence and arousal ratings of words: a population-based study.
TL;DR: The aim was to assess the feasibility of obtaining and determining the structure of valence and arousal ratings in the setting of the large observational BiDirect Study, and the roles of age, sex, depression, and anxiety for valenceand arousal ratings of words were explored.
Neurofilament L gene is not a genetic factor of sporadic and familial Parkinson’s disease
TL;DR: The role of neurofilament light (NF-L), another component of LB aggregation, is investigated to gain further insight into the pathogenesis of PD, finding no significant differences between PD patients and 344 control individuals.
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