Kennedy R. Lees
University of Glasgow
467 Papers
3.9K Citations
Kennedy R. Lees is an academic researcher from University of Glasgow. The author has contributed to research in topics: Stroke & Medicine. The author has an hindex of 86, co-authored 455 publications. Previous affiliations of Kennedy R. Lees include Ludwig Maximilian University of Munich & Cedars-Sinai Medical Center.
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Papers
•Journal Article
Abstract 2481: Day 7 NIHSS is a Sensitive Outcome Measure for Exploratory Clinical Trials in Acute Stroke: Evidence from the Virtual International Stroke Trials Archive (VISTA)
TL;DR: 7 day NIHSS score appears a sensitive endpoint that should be validated in a randomised trial dataset for use in exploratory stroke trials.
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Systemic and cerebral hemodynamic responses to the noncompetitive N-methyl-D-aspartate (NMDA) antagonist CNS 1102
TL;DR: CNS 1102 administration was associated with light-headedness, mild disorientation, perioral and peripheral paresthesias, and flushing, but in the presence of systemic hypertension, adverse cerebral hemodynamic changes were not observed.
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Clinically important atrial arrhythmia and stroke risk: a UK-wide online survey among stroke physicians and cardiologists
TL;DR: There is a lack of consensus on the diagnosis and management of patients with brief runs of atrial arrhythmia detected on ambulatory ECG and further research is needed to clarify the risk of stroke in this unique population of patients.
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Advances in neuroprotection trials.
TL;DR: Recent trials have not yet identified a neuroprotectant, but have greatly assisted the prospects of success and utilisation of existing trial databases can enhance statistical approaches to outcome assessment.
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•Journal Article
Role of angiotensin in the extravascular system.
TL;DR: Angiotensin receptors are present in many tissue types, including adrenal cortex, renal glomeruli, heart, hypothalamus, liver, pancreas, pituitary, platelets, renal tubules, uterus and vascular smooth muscle and high-affinity receptor subtypes have been identified by radioligand binding with antagonists.
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