Kengo Furuichi
Kanazawa University
166 Papers
1K Citations
Kengo Furuichi is an academic researcher from Kanazawa University. The author has contributed to research in topics: Medicine & Kidney. The author has an hindex of 40, co-authored 135 publications. Previous affiliations of Kengo Furuichi include Kanazawa Medical University.
Chat about Author
Papers
Fibrocytes are involved in the pathogenesis of human chronic kidney disease
Norihiko Sakai,Kengo Furuichi,Yasuyuki Shinozaki,Hiroyuki Yamauchi,Tadashi Toyama,Shinji Kitajima,Toshiya Okumura,Satoshi Kokubo,Motoo Kobayashi,Kazuya Takasawa,Shin'ichi Takeda,Mitsuhiro Yoshimura,Shuichi Kaneko,Takashi Wada +13 more
TL;DR: The results suggest that fibrocytes may be involved in the pathogenesis of chronic kidney disease through the interaction with macrophages as well as CCL2, which was significantly decreased during convalescence induced by glucocorticoid therapy.
46
Glomerular ICAM-1 Expression Related to Circulating TNF-α in Human Glomerulonephritis
Hitoshi Yokoyama,Masayoshi Takaeda,Takashi Wada,Satoshi Ohta,Yukimasa Hisada,Chikako Segawa,Kengo Furuichi,Kenichi Kobayashi +7 more
TL;DR: To clarify the in vivo involvement of cellular adhesion molecules and cytokines in human glomerulonephritis, the glomerular and interstitial expression of intercellular adhesion molecule and cytokine expression is investigated.
44
Clinical significance of urinary liver-type fatty acid-binding protein as a predictor of ESRD and CVD in patients with CKD
Katsuomi Matsui,Atsuko Kamijo-Ikemori,Naohiko Imai,Takeshi Sugaya,Takashi Yasuda,Shinobu Tatsunami,Tadashi Toyama,Miho Shimizu,Kengo Furuichi,Takashi Wada,Yugo Shibagaki,Kenjiro Kimura +11 more
TL;DR: Urinary L-FABP may be a useful prognostic marker of progression to ESRD and the onset of CVD in patients with CKD.
43
Involvement of bone-marrow-derived cells in kidney fibrosis
TL;DR: Bone-marrow-derived cells positive for CD45 or CD34, and type 1 (pro)collagen dependent on the chemokine and renin–angiotensin systems migrate into diseased kidneys and enhance synthesis matrix protein, cytokines/chemokines, and profibrotic growth factors, which may promote and escalate chronic inflammatory processes and possible interaction with resident stromal cells, thereby perpetuating kidney fibrosis.
Combined pure red cell aplasia and autoimmune hemolytic anemia in systemic lupus erythematosus with anti-erythropoietin autoantibodies.
Akinori Hara,Takashi Wada,Shinji Kitajima,Tadashi Toyama,Toshiya Okumura,Kiyoki Kitagawa,Yasunori Iwata,Norihiko Sakai,Kengo Furuichi,Masato Higuchi,Shuichi Kaneko +10 more
TL;DR: A 42‐year‐old woman with systemic lupus erythematosus was admitted to the authors' hospital because of severe anemia and radioimmunoprecipitation assay revealed that her serum was positive for anti‐erythropoietin antibodies before therapy, and immunosuppressive treatment improved the anemia accompanied with disappearance of the autoantibodies.
38