Keith J. Peevy
University of South Alabama
41 Papers
432 Citations
Keith J. Peevy is an academic researcher from University of South Alabama. The author has contributed to research in topics: Vascular smooth muscle & Gestational age. The author has an hindex of 15, co-authored 41 publications. Previous affiliations of Keith J. Peevy include University of Medicine and Dentistry of New Jersey.
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Papers
Chest wall restriction limits high airway pressure-induced lung injury in young rabbits
TL;DR: Data indicate that volume distension of the lung rather than high PIP per se produces microvascular damage in the immature rabbit lung, which is in line with previous work on tidal volume and direct pressure effects.
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Lung Edema Caused by High Peak Inspiratory Pressures in Dogs: Role of Increased Microvascular Filtration Pressure and Permeability
James C. Parker,Lucrecia A. Hernandez,Gesina L. Longenecker,Keith J. Peevy,Walter H. Johnson +4 more
TL;DR: Lymph prostacyclin concentration in pulmonary lymph, measured as the stable metabolite 6-0-PGF1 alpha, was increased significantly by 70 to 150% in both groups during the periods of increased Paw and increased PLA, but it was not significantly different between the groups.
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•Journal Article
Epidemiology of inguinal hernia in preterm neonates.
TL;DR: The data demonstrate a previously unrecognized association between neonatal inguinal hernia and intrauterine growth retardation among infants less than or equal to 32 weeks' gestational age, which significantly increases the risk for development of ingUinal hernias.
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Barotrauma and microvascular injury in lungs of nonadult rabbits: effect of ventilation pattern.
TL;DR: The data indicate that high PIP, and to a lesser extent, high gas flow rates cause microvascular injury in the compliant nonadult lung and suggest that the combination of highPIP and high gas Flow rates are the most threatening to microv vascular integrity.
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Release of norepinephrine from adrenergic nerve endings of blood vessels is modulated by endothelium-derived relaxing factor
TL;DR: The findings support the conclusion that the endothelium can inhibit release of NE from sympathetic nerve innervating canine pulmonary artery and vein and act as an endogenous inhibitor or sympathetic neurotransmitter release.
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