8 Papers
13 Citations
Ke Ma is an academic researcher from University Health Network. The author has contributed to research in topics: Neurotransmission & Long-term potentiation. The author has an hindex of 3, co-authored 5 publications. Previous affiliations of Ke Ma include Jilin University & University of Toronto.
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Papers
Crucial Role of Postsynaptic Syntaxin 4 in Mediating Basal Neurotransmission and Synaptic Plasticity in Hippocampal CA1 Neurons.
Na-Ryum Bin,Na-Ryum Bin,Ke Ma,Ke Ma,Hidekiyo Harada,Chi-Wei Tien,Fiona Bergin,Fiona Bergin,Kyoko Sugita,Thomas T. Luyben,Thomas T. Luyben,Masahiro Narimatsu,Zhengping Jia,Jeffrey L. Wrana,Jeffrey L. Wrana,Philippe P. Monnier,Philippe P. Monnier,Liang Zhang,Liang Zhang,Kenichi Okamoto,Kenichi Okamoto,Shuzo Sugita,Shuzo Sugita +22 more
TL;DR: The data reveal a crucial role of syntaxin 4 in trafficking of ionotropic glutamate receptors that are essential for basal neurotransmission, synaptic plasticity, and spatial memory.
25
C2 Domains of Munc13-4 Are Crucial for Ca2+-Dependent Degranulation and Cytotoxicity in NK Cells.
Na Ryum Bin,Na Ryum Bin,Ke Ma,Ke Ma,Chi Wei Tien,Chi Wei Tien,Siyan Wang,Siyan Wang,Dan Zhu,Seungmee Park,Seungmee Park,Ekaterina Turlova,Kyoko Sugita,Ryutaro Shirakawa,Peter van der Sluijs,Hisanori Horiuchi,Hong Shuo Sun,Philippe P. Monnier,Philippe P. Monnier,Herbert Y. Gaisano,Shuzo Sugita,Shuzo Sugita +21 more
TL;DR: It is demonstrated that both C2 domains of Munc13-4 play critical roles in Ca2+-dependent exocytosis and cytotoxicity by regulating single-granule membrane fusion dynamics in immune cells.
18
Syntaxin-3 is dispensable for basal neurotransmission and synaptic plasticity in postsynaptic hippocampal CA1 neurons.
Shan Shi,Ke Ma,Ke Ma,Na Ryum Bin,Hidekiyo Harada,Xiaoyu Xie,Xiaoyu Xie,Mengjia Huang,Mengjia Huang,Haiyu Liu,Haiyu Liu,Soomin Lee,Soomin Lee,Xue Fan Wang,Roberto Adachi,Philippe P. Monnier,Philippe P. Monnier,Liang Zhang,Liang Zhang,Shuzo Sugita,Shuzo Sugita +20 more
TL;DR: The data suggest that syntaxin-3 is dispensable for hippocampal basal neurotransmission and synaptic plasticity, and further supports the notion that Syntaxin-4 is the major isoform mediating these processes.
Neuronal SNAP-23 is critical for synaptic plasticity and spatial memory independently of NMDA receptor regulation
Mengjia Huang,Na-Ryum Bin,Jayant Rai,Ke Ma,C. Chow,Sarah Eide,Hidekiyo Harada,Jianbing Xiao,Daorong Feng,Hong-Shuo Sun,Zhong-Ping Feng,Herbert Y. Gaisano,Jeffrey E. Pessin,Philippe P. Monnier,Ken-ichi Okamoto,L. Zhang,Shuzo Sugita +16 more
TL;DR: In this paper , the role of neuronal synaptosomal-associated protein-23 (SNAP-23) by analyzing pyramidal-neuron specific SNAP-23 conditional knockout (cKO) mice was investigated.
5
Observations From a Mouse Model of Forebrain Voa1 Knockout: Focus on Hippocampal Structure and Function.
Ke Ma,Ke Ma,Na-Ryum Bin,Shan Shi,Shan Shi,Hidekiyo Harada,Yoh Wada,Ge-Hong-Sun Wada,Philippe P. Monnier,Philippe P. Monnier,Shuzo Sugita,Shuzo Sugita,Liang Zhang,Liang Zhang +13 more
TL;DR: It is postulate that the Voa1 knockout mice may serve as a valuable model for further investigation of V-ATPase dysfunction related neuronal degeneration and functional abnormalities in forebrain areas particularly the hippocampus.