Katrine Frønsdal
University of Oslo
10 Papers
72 Citations
Katrine Frønsdal is an academic researcher from University of Oslo. The author has contributed to research in topics: LNCaP & Cell. The author has an hindex of 8, co-authored 8 publications.
Chat about Author
Papers
In vitro expansion of human mesenchymal stem cells: choice of serum is a determinant of cell proliferation, differentiation, gene expression, and transcriptome stability.
TL;DR: It is discovered that hMSCs may be expanded rapidly and with stable gene expression in AS in the absence of growth factors, whereas FBS induces a more differentiated and less stable transcriptional profile.
506
Isolation and transcription profiling of purified uncultured human stromal stem cells: alteration of gene expression after in vitro cell culture.
Andrew C. Boquest,Aboulghassem Shahdadfar,Katrine Frønsdal,Olafur E. Sigurjonsson,Siv H. Tunheim,Philippe Collas,Jan E. Brinchmann +6 more
TL;DR: Two populations of CD45-CD34+CD105+ cells from human adipose tissue which could be separated based on expression of CD31 have stromal stem cell properties which may make them useful for tissue engineering.
373
CREB binding protein is a coactivator for the androgen receptor and mediates cross-talk with AP-1.
TL;DR: CBP significantly increased the ability of endogenous AR in LNCaP cells to activate transcription from an AR-dependent reporter construct and that the transcriptional interference between AR and AP-1 is the result of competition for limiting amounts of CBP in the cell.
235
Histone deacetylase inhibitors differentially mediate apoptosis in prostate cancer cells
TL;DR: In this paper, three structurally unrelated histone deacetylase (HDAC) inhibitors, trichostatin A (TSA), depsipeptide (FR901228), and sodium butyrate, were evaluated in the prostate cancer cell lines LNCaP, DU-145, and PC-3.
62
DNA binding-independent transcriptional activation by the androgen receptor through triggering of coactivators.
TL;DR: A novel ligand-dependent transactivation function for AR that is independent of its DNA binding ability is described, which suggests that triggering may constitute an important part of the mechanism by which AR regulates transcription.
24