Katrina E. Traber
Boston University
29 Papers
172 Citations
Katrina E. Traber is an academic researcher from Boston University. The author has contributed to research in topics: Lung & Pneumonia. The author has an hindex of 11, co-authored 17 publications. Previous affiliations of Katrina E. Traber include University of California, Berkeley & New York University.
Chat about Author
Papers
Differentiation of Human Pluripotent Stem Cells into Functional Lung Alveolar Epithelial Cells.
Anjali Jacob,Anjali Jacob,Michael Morley,Finn Hawkins,Finn Hawkins,Katherine B. McCauley,Katherine B. McCauley,J. C. Jean,J. C. Jean,Hillary B. Heins,Cheng-Lun Na,Timothy E. Weaver,Marall Vedaie,Marall Vedaie,Killian Hurley,Killian Hurley,Anne Hinds,Scott J. Russo,Seunghyi Kook,William J. Zacharias,Matthias Ochs,Katrina E. Traber,Lee J. Quinton,Ana M. Crane,Brian R. Davis,Frances V. White,Jennifer A. Wambach,Jeffrey A. Whitsett,F. Sessions Cole,Edward E. Morrisey,Susan H. Guttentag,Michael F. Beers,Darrell N. Kotton,Darrell N. Kotton +33 more
TL;DR: PSC-derived AEC2s provide a platform for disease modeling and future functional regeneration of the distal lung, and exhibit self-renewal capacity, and display additional A EC2 functional capacities.
504
Agr Function in Clinical Staphylococcus Aureus Isolates
Katrina E. Traber,Elsie Lee,Sarah A. Benson,Rebecca M. Corrigan,Mariela Cantera,Bo Shopsin,Richard P. Novick +6 more
TL;DR: It is indicated that while most clinical isolates are haemolytic and agr(+), non-haemolytics and agR(-) strains are found in S. aureus infections, and that agr (+) and agg(-) variants may have a cooperative interaction in certain types of infections.
358
A slipped-mispairing mutation in AgrA of laboratory strains and clinical isolates results in delayed activation of agr and failure to translate delta- and alpha-haemolysins.
TL;DR: It is shown here that the non‐haemolytic phenotype of RN4220 is caused by an extra A residue in a run of seven As at the 3′ end of agrA (agrA‐8A), which causes a frameshift that results in the addition of three amino acyl residues to the C‐terminal end of the protein.
136
The agr Radiation: an Early Event in the Evolution of Staphylococci
Jesse S. Wright,Katrina E. Traber,Rebecca M. Corrigan,Sarah A. Benson,James M. Musser,James M. Musser,Richard P. Novick +6 more
TL;DR: Evidence is presented, using a newly developed, luciferase-based agr typing scheme, that the evolutionary divergence of the agr system was an early event in the evolution of the staphylococci and long preceded the development of the nucleotide polymorphisms presently used for genotyping.
133
Inhibition of nf-kappa b activation in human t-cell lines by anetholdithiolthione
TL;DR: ADT, a pro-glutathione antioxidant, was observed to be capable of modulating NF-κB activation and had significantly higher glutathione levels as compared with untreated cells.
65