Kana Miyamoto
Keio University
64 Papers
206 Citations
Kana Miyamoto is an academic researcher from Keio University. The author has contributed to research in topics: Osteoclast & Osteoporosis. The author has an hindex of 26, co-authored 56 publications. Previous affiliations of Kana Miyamoto include Toho University & Kumamoto University.
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Papers
An ionic silver coating prevents implant-associated infection by anaerobic bacteria in vitro and in vivo in mice
Tomoya Soma,Ryotaro Iwasaki,Yuiko Sato,Tami Kobayashi,Eriko Ito,Tatsuaki Matsumoto,Atsushi Kimura,F. Homma,Keitarou Saiki,Yukihiro Takahashi,Kana Miyamoto,Morio Matsumoto,Masaya Nakamura,Mayu Morita,Ken Ishii,Seiji Asoda,Hiromasa Kawana,Zhu Xingyu,Mamoru Aizawa,Taneaki Nakagawa,Takeshi Miyamoto +20 more
TL;DR: In this paper , the authors show antibacterial activity of silver ions coated on titanium rods, even against the anaerobic bacteria Porphyromonas gingivalis (P. gingfish), both in vitro and in vivo.
FoxO3a regulates hematopoietic homeostasis through a negative feedback pathway in conditions of stress or aging
TL;DR: It is reported that mice lacking the transcription factor forkhead box O3a (FoxO3a) develop neutrophilia associated with inhibition of the up-regulation of negative regulator of cell proliferation, Sprouty-related Ena/VASP homology 1 domain-containing proteins 2 (Spred2) and AKT and ERK activation, in HSCs during hematopoietic recovery following myelosuppressive stress conditions.
CD24 is expressed specifically in the nucleus pulposus of intervertebral discs.
Nobuyuki Fujita,Takeshi Miyamoto,Jun-ichi Imai,Naobumi Hosogane,Toru Suzuki,Mitsuru Yagi,Kozo Morita,Ken Ninomiya,Kana Miyamoto,Hironari Takaishi,Morio Matsumoto,Hideo Morioka,Hiroo Yabe,Kazuhiro Chiba,Shinya Watanabe,Yoshiaki Toyama,Toshio Suda +16 more
TL;DR: CD24, a glycosylphosphatidylinositol anchor protein, was identified as a cell surface marker for NP and was detected in the herniated NP and chordoma, a malignant primary tumor derived from notochordal cells, while it was absent in chondrosarcoma.
Interleukin-1 Receptor-associated Kinase-4 (IRAK4) Promotes Inflammatory Osteolysis by Activating Osteoclasts and Inhibiting Formation of Foreign Body Giant Cells
Eri Katsuyama,Hiroya Miyamoto,Tami Kobayashi,Yuiko Sato,Wu Hao,Hiroya Kanagawa,Atsuhiro Fujie,Toshimi Tando,Ryuichi Watanabe,Mayu Morita,Kana Miyamoto,Yasuo Niki,Hideo Morioka,Morio Matsumoto,Yoshiaki Toyama,Takeshi Miyamoto +15 more
TL;DR: It is demonstrated that osteoclasts and FBGCs are reciprocally regulated and identified IRAK4 as a potential therapeutic target to inhibit stimulated osteoclastogenesis and rescue inhibited FBGC formation under inflammatory and infectious conditions without altering physiological bone resorption.