K. V. Yatsenko
8 Papers
11 Citations
K. V. Yatsenko is an academic researcher. The author has contributed to research in topics: Periventricular leukomalacia & Adipose tissue. The author has an hindex of 2, co-authored 8 publications.
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Papers
Ultrastructural study of mouse adipose-derived stromal cells induced towards osteogenic direction.
O. M. Tsupykov,Alina Ustymenko,V. M. Kyryk,Ekaterina Smozhanik,K. V. Yatsenko,G. M. Butenko,Galina Skibo +6 more
TL;DR: At the end of the osteogenic differentiation, the ASCs transformed from their original fibroblast‐like appearance to having a polygonal osteoblast‐like morphology, providing the ultrastructural basis for further studies on the cellular mechanisms involved in osteogenic differentiated cells.
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A novel model of periventricular leukomalacia on mouse organotypic brain slice culture
O. M. Tsupykov,I. V. Lushnikova,Y. A. Nikandrova,K. V. Yatsenko,Alina Ustymenko,V. M. Kyryk,G. M. Butenko,G. G. Skybo +7 more
- 30 Nov 2016
TL;DR: The authors' model of PVL developed on cultured organotypic mice brain slices is novel and promising tool to study pathogenic mechanisms of cerebral white matter lesions and ways of neuroprotection in this pathology, including pharmacological agents and transplantation of stem cells.
3D culture of murine adipose-derived multipotent mesenchymal stromal cells in hydrogel based on carbomer 974P
Vitalii Kyryk,O. Kuchuk,A. Mamchur,Alina Ustymenko,T. Lutsenko,O. M. Tsupykov,K. V. Yatsenko,Galina Skibo,N. Bilko,D. Bilko +9 more
- 30 Nov 2018
TL;DR: Three-dimensional hydrogel based on carbomer 974P is capable to support cells, provide the necessary cytoarchitectonics, maintain intercellular interactions, which can promote further long-term survival and specialization of graft.
Adipose-Derived Stem Cells Reduce Lipopolysaccharide-Induced Myelin Degradation and Neuroinflammatory Responses of Glial Cells in Mice.
K. V. Yatsenko,Iryna Lushnikova,Alina Ustymenko,M. A. Patseva,I. O. Govbakh,Vitalii Kyryk,O. M. Tsupykov +6 more
TL;DR: It was found that LPS caused micro- and astroglial activation and oligodendrocyte degradation, whereas the presence of ASCs significantly reduced the damaging effects, and it should be noted that the observed ASCs protection in a non-contact co-culture suggested that this effect was due to humoral factors via ASC-released biomodulatory molecules.
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