Junli Liu
Chinese Academy of Sciences
6 Papers
8 Citations
Junli Liu is an academic researcher from Chinese Academy of Sciences. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 5, co-authored 6 publications.
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Papers
Macrophage/microglial Ezh2 facilitates autoimmune inflammation through inhibition of Socs3
Xingli Zhang,Yan Wang,Jia Yuan,Ni Li,Siyu Pei,Jing Xu,Xuan Luo,Chaoming Mao,Junli Liu,Tao Yu,Shucheng Gan,Qianqian Zheng,Yinming Liang,Weixiang Guo,Ju Qiu,Gabriela Constantin,Jin Jin,Jun Qin,Yichuan Xiao +18 more
TL;DR: Ezh2 is established as a macrophage lineage-specific mediator of autoimmune inflammation and highlight a previously unknown mechanism of Ezh2 function.
The ubiquitin ligase Peli1 inhibits ICOS and thereby Tfh-mediated immunity.
Xinfang Huang,Shumeng Hao,Junli Liu,Yuanyuan Huang,Manman Liu,Chunyuan Xiao,Yan Wang,Siyu Pei,Tao Yu,Jing Xu,Haikun Wang,Dongfang Dai,Xiao Su,Yichuan Xiao +13 more
TL;DR: In this article, the authors identified Peli1, a T cell-enriched E3 ubiquitin ligase, as an intrinsic regulator that inhibits T follicular helper (Tfh) cell differentiation.
Higher activation of the interferon-gamma signaling pathway in systemic lupus erythematosus patients with a high type I IFN score: relation to disease activity.
Manman Liu,Junli Liu,Shumeng Hao,Ping Wu,Xiaoyan Zhang,Yichuan Xiao,Gengru Jiang,Xinfang Huang +7 more
TL;DR: The relationship among IFN-γ, type I interferons (IFNs) and clinical features have not been extensively studied as mentioned in this paper, however, the relationship between IFNγ levels and type I IFN scores has been extensively investigated.
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Activation status of CD56dim natural killer cells is associated with disease activity of patients with systemic lupus erythematosus
Manman Liu,Manman Liu,Junli Liu,Xiaoyan Zhang,Yichuan Xiao,Gengru Jiang,Xinfang Huang,Xinfang Huang +7 more
TL;DR: In patients with SLE, the proportion of CD56 dim NK cells showed a decreased trend and CD56Dim NK cells were phenotypically activated which partially reflects the disease activity.
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Tpl2 Protects Against Fulminant Hepatitis Through Mobilization of Myeloid-Derived Suppressor Cells.
Jing Xu,Siyu Pei,Yan Wang,Junli Liu,Youcun Qian,Mingzhu Huang,Yanyun Zhang,Yanyun Zhang,Yichuan Xiao +8 more
TL;DR: Increased MDSC in the inflamed liver specifically restrained the local proliferation of infiltrated pathogenic CD4+ T cells, and thus protected against the inflammation-induced acute liver failure.