73 Papers
138 Citations
Jun Yin is an academic researcher from Shanghai Jiao Tong University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 20, co-authored 63 publications. Previous affiliations of Jun Yin include Pennington Biomedical Research Center & Ruijin Hospital.
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Papers
Butyrate Improves Insulin Sensitivity and Increases Energy Expenditure in Mice
Zhanguo Gao,Jun Yin,Jin Jin Zhang,Robert Ward,Roy J. Martin,Michael Lefevre,William T. Cefalu,Jianping Ye +7 more
TL;DR: Dietary supplementation of butyrate can prevent and treat diet-induced insulin resistance in mouse and the mechanism ofbutyrate action is related to promotion of energy expenditure and induction of mitochondria function.
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Hypoxia is a potential risk factor for chronic inflammation and adiponectin reduction in adipose tissue of ob/ob and dietary obese mice
TL;DR: Experimental evidence thatHypoxia occurs in adipose tissue in obese mice and that adipose hypoxia may contribute to the endocrine alterations is provided and a potential role of hypoxIA in the induction of chronic inflammation and inhibition of adiponectin in the adipose tissues in obesity is suggested.
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Efficacy of berberine in patients with type 2 diabetes mellitus
Jun Yin,Huili Xing,Jianping Ye +2 more
TL;DR: It is indicated that berberine is a potent oral hypoglycemic agent with beneficial effects on lipid metabolism and homeostasis model assessment of insulin resistance index in type 2 diabetes mellitus patients.
Berberine Improves Glucose Metabolism through Induction of Glycolysis
TL;DR: Berberine-induced AMPK activation is likely a consequence of mitochondria inhibition that increases the AMP/ATP ratio and reduction in oxygen consumption, which is related to inhibition of glucose oxidation in mitochondria.
Role of hypoxia in obesity-induced disorders of glucose and lipid metabolism in adipose tissue
TL;DR: The data suggest that ATH may promote FFA release and inhibit glucose uptake in adipocytes by inhibition of the insulin-signaling pathway and induction of cell death in ob/ob mice.
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